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Intracerebral Hemorrhage clinical trials

View clinical trials related to Intracerebral Hemorrhage.

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NCT ID: NCT02866838 Completed - Clinical trials for Intracerebral Hemorrhage

Treatment of Intracerebral Hemorrhage in Patients on Non-vitamin K Antagonist

TICH-NOAC
Start date: December 2016
Phase: Phase 2/Phase 3
Study type: Interventional

Novel, non-vitamin K antagonist oral anticoagulants (NOAC) target selected players in the coagulation cascade as the direct thrombin inhibitor dabigatran and the factor Xa-inhibitors apixaban and rivaroxaban. Intracerebral hemorrhage (ICH) is the most feared complication of NOAC treatment (NOAC-ICH). Outcome of NOAC-ICH can be devastating and is a major cause of death and disability. There is no proven treatment for NOAC-ICH. Hematoma expansion (HE) is associated with unfavorable outcome. Tranexamic acid (TA) is an anti-fibrinolytic drug that is used in a number of bleeding conditions other than ICH.

NCT ID: NCT02567162 Completed - Clinical trials for Intracerebral Hemorrhage

Multinational Survey of Epidemiology, Morbidity, and Outcomes in Intracerebral Hemorrhage

MNEMONICH
Start date: November 2015
Phase: N/A
Study type: Observational [Patient Registry]

The aim of this project is development of a comprehensive, prospective, multinational, multicenter, hospital-based, data repository for patients with intracerebral hemorrhage.

NCT ID: NCT02533960 Completed - Clinical trials for Cardiovascular Diseases

Registry of Acute Stroke Under Novel Oral Anticoagulants - Prime

RASUNOA-Prime
Start date: June 2015
Phase:
Study type: Observational

The Registry of Acute Stroke Under Novel Oral Anticoagulants-Prime (RASUNOA-Prime), an investigator-initiated study, is a German multicenter, prospective, observational registry. It is performed at about 50 certified stroke-units and supported by an unrestricted grant from different pharmaceutical companies to the Heidelberg University Hospital. RASUNOA-Prime is designed to assess the emergency management of acute ischemic and hemorrhagic stroke patients with atrial fibrillation (AF) under different anticoagulation schemes pre stroke: Non-vitamin K antagonist oral anticoagulants (NOAC), Vitamin K antagonists (VKA), and no anticoagulation.

NCT ID: NCT02478177 Completed - Stroke Clinical Trials

Addressing Real-world Anticoagulant Management Issues in Stroke

ARAMIS
Start date: September 2015
Phase:
Study type: Observational [Patient Registry]

The Addressing Real-world Anticoagulant Management Issues in Stroke (ARAMIS) registry is designed to provide important and timely insight into the management of acute stroke patients who are on novel oral anticoagulants in community practice.

NCT ID: NCT02175225 Completed - Clinical trials for Intracerebral Hemorrhage

Intracerebral Hemorrhage Deferoxamine Trial - iDEF Ttrial

Start date: October 2014
Phase: Phase 2
Study type: Interventional

The investigators hypothesize that treatment with the iron chelator, Deferoxamine Mesylate, improves the outcome of patients with brain hemorrhage. The purpose of this study is to determine whether treatment with Deferoxamine Mesylate is of sufficient promise to improve outcome before pursuing a larger clinical trial to examine its effectiveness as a treatment for intracerebral hemorrhage.

NCT ID: NCT02078037 Completed - Clinical trials for Intracerebral Hemorrhage

Systemic Normothermia in Intracerebral Hemorrhage (ICH)

SNICH
Start date: March 2014
Phase: Phase 0
Study type: Interventional

Patients with a supratentorial intracerebral hemorrhage will be randomly assigned to either the experimental group which will keep them at a normal body temperature or the standard of care group. The investigators propose to test the hypothesis that prophylactic forced normothermia in patients with ICH leads to less systemic inflammation and decreased perihematomal edema.

NCT ID: NCT01971359 Completed - Clinical trials for Intracerebral Hemorrhage

Clinical Outcomes Following Parafascicular Surgical Evacuation of Intracerebral Hemorrhage: A Pilot Study

MISPACE
Start date: September 2013
Phase: N/A
Study type: Observational

This study is determining the clinical outcomes, based on neurological testing, for the parafascicular minimally invasive clot evacuation technique. We will collect data from the time of surgery up until 90 days post procedure. We will also be looking at financial data as well. - H(0): there is no economic benefit to the system with early surgical intervention for ICH - Alternative Hypothesis:H(1) Assuming clinical equipoise, i.e., no benefit in clinical outcome with early surgical intervention (null hypotheses) H(0) is correct), the ability to accelerate the patients care from the entry point to the exit point will result in a significant economic advantage to the system through cost reduction.

NCT ID: NCT01827046 Completed - Clinical trials for Intracerebral Hemorrhage

Minimally Invasive Surgery Plus Rt-PA for ICH Evacuation Phase III

MISTIE-III
Start date: December 30, 2013
Phase: Phase 3
Study type: Interventional

A phase III, randomized, case-controlled, open-label, 500-subject clinical trial of minimally invasive surgery plus rt-PA in the treatment of intracerebral hemorrhage (ICH).

NCT ID: NCT01805895 Completed - Clinical trials for Intracerebral Hemorrhage

A Pilot Study of Minocycline in Intracerebral Hemorrhage Patients

MACH
Start date: February 2013
Phase: Phase 1/Phase 2
Study type: Interventional

The MACH Trial is a pilot study of 400mg minocycline over five days in acute intracerebral hemorrhage patients. The study will evaluation the safety and efficacy of minocycline in intracerebral hemorrhage patients.

NCT ID: NCT01619709 Completed - Clinical trials for Intracerebral Hemorrhage

Amyloid Imaging and Cognitive Impairment After Intracerebral Hemorrhage

COGHIC-AV45
Start date: January 2012
Phase: N/A
Study type: Interventional

To evaluate Pet AV-45 Amyloid imaging in the etiological diagnosis of primary non traumatic intracerebral hemorrhage (Cerebral Amyloid Angiopathy and hypertension related hemorrhage).We hypothesize that patients with lobar hemorrhage (probably related to Cerebral Amyloid Angiopathy) will have a greater AV45 cortical binding than patients with deep hemorrhage (probably related to hypertension).