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Cerebral Vasospasm clinical trials

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NCT ID: NCT00839449 Completed - Clinical trials for Subarachnoid Hemorrhage

Eicosapentaenoic Acid Cerebral Vasospasm Therapy Study

EVAS
Start date: December 2004
Phase: Phase 4
Study type: Interventional

Cerebral vasospasm following subarachnoid hemorrhage (SAH) is the most common cause of morbidity and mortality. Recent studies indicate that Rho-kinase play an important role in the occurrence of such cerebral vasospasm. Eicosapentaenoic acid (EPA) inhibits sphingosylphosphorylcholine (SPC)-induced Rho-kinase activation in vitro. So this study examines whether EPA prevents cerebral vasospasm occurrence after SAH in patients.

NCT ID: NCT00692744 Completed - Hydrocephalus Clinical Trials

Quality of Life in Elderly After Aneurysmal Subarachnoid Hemorrhage (SAH)

FASHE
Start date: October 2008
Phase: N/A
Study type: Observational

In all the Western populations, the annual incidence of subarachnoid hemorrhage (SAH) increase with age. In patients older than 70 years, the occurrence of SAH exposes them to high risk of morbidity and a poor quality of life. In this age bracket, the single randomized which compared endovascular coiling to microsurgical clipping (ISAT Study) showed that the relative risk of morbidity increased after coiling. Moreover, some prospectives studies about endovascular coiling described favorable outcome in 48% to 63% of patients, complete occlusion in 51% to 69% and a procedural complication rate in 13% to 19%. From prospectives series, the proportion of favorable outcome after microsurgical clipping was estimated around 66% but the procedural complications are few reported. The outcome for patients treated conservatively was catastrophic. Lastly, the hydrocephalus in this age class is common, occurring in 55% of patients. The study hypothesis is that, in this age class, no difference exists between the 2 obliteration procedures. An accurate evaluation of result in term of functional disability, quality of life and prognosis predictive factors seems a judicious question.

NCT ID: NCT00585559 Terminated - Clinical trials for Aneurysmal Subarachnoid Hemorrhage

Acetaminophen in aSAH to Inhibit Lipid Peroxidation and Cerebral Vasospasm

Start date: April 2007
Phase: Phase 3
Study type: Interventional

The objective of this study is to determine whether acetaminophen (APAP), N-acetylcysteine (NAC), and APAP in combination with NAC will inhibit lipid peroxidation in aneurysmal subarachnoid hemorrhage (aSAH), utilizing F2-IsoPs as biomarkers for lipid peroxidation.

NCT ID: NCT00582868 Terminated - Clinical trials for Subarachnoid Hemorrhage

Use of Brain Oxygen Tension Level and Cleaved-tau Protein to Detect Vasospasm After SAH

Start date: May 2007
Phase: N/A
Study type: Observational

The purpose of this study is to investigate if brain oxygen levels, levels of a specific protein in the cerebrospinal fluid and blood (Cleaved-tau protein), and brain blood flow can predict spasm of brain blood vessels after bleeding in the brain from a ruptured aneurysm.

NCT ID: NCT00487461 Terminated - Clinical trials for Aneurysmal Subarachnoid Hemorrhage

Use of Simvastatin for the Prevention of Vasospasm in Aneurysmal Subarachnoid Hemorrhage

Start date: May 2007
Phase: N/A
Study type: Interventional

The purpose of this study is to test whether treatment with a drug called Simvastatin prevents and improves outcome in patients who have Subarachnoid bleeding. Simvastatin is currently approved for the treatment of high cholesterol levels.