Atrial Fibrillation Clinical Trial
Atrial fibrillation (AF) is the most common cardiac arrhythmia. Because of its major impact
on the general morbidity and risk of stroke, AF is a great concern for public health. Several
mechanisms, including endothelial dysfunction and inflammatory processes, have been
postulated as predisposing factors for AF, as well as for stroke.
Both clinical and experimental studies highlight inflammation as a predisposing factor for AF
and its complications. Nevertheless, the source of high inflammatory proteins in patients
with AF is still unknown. We hypothesized that multilevel intracardiac and extracardiac (left
femoral vein, coronary sinus, left atrium, pulmonary vein) measurements of several
inflammatory proteins (VEGF) would help assessing the extent and the source of inflammation
in AF patients.
The measurement of von Willebrand factor (vWF) levels in multiple vascular sites would also
help to define the site of endothelial dysfunction and of production of this thrombogenic
factor.
Although AF is associated with an increased risk of stroke, the risk is not homogeneous.
Permanent and persistent AF are associated with similar thromboembolic risk to that of
paroxysmal AF.
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