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Metabolic Diseases clinical trials

View clinical trials related to Metabolic Diseases.

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NCT ID: NCT03151239 Completed - Clinical trials for Glucose Metabolism Disorders

Effect of "Nicotinamide Mononucleotide" (NMN) on Cardiometabolic Function

NMN
Start date: July 1, 2017
Phase: N/A
Study type: Interventional

The purpose of the study is to understand the effect of the dietary supplement "Nicotinamide mononucleotide" on metabolic health in people.

NCT ID: NCT03133741 Completed - Clinical trials for Glucose Metabolism Disorders

Inhibition of the Endogenous GIP Response With a GIP Receptor Antagonist

GA-3
Start date: March 1, 2017
Phase: N/A
Study type: Interventional

Delinieation of GIP's effects during an oral glucose tolerance test (OGTT) in humans using GIP receptor antagonisation.

NCT ID: NCT03125356 Completed - Clinical trials for Nutritional and Metabolic Diseases

Low-calorie Sweeteners and Adipose Signaling

Start date: January 1, 2017
Phase: N/A
Study type: Interventional

The purpose of this study is to determine whether eight weeks of low-calorie sweetener (LCS) exposure exerts cardiovascular and/or metabolic effects among LCS-naïve overweight and obese adolescents and young adults. We will examine changes in gene expression in subcutaneous fat and will correlate these molecular changes with plasma biochemistry.

NCT ID: NCT03119350 Completed - Obesity Clinical Trials

Mitochondrial Energy Metabolism in Obese Women

Start date: April 1, 2016
Phase: N/A
Study type: Interventional

Considering that the failure of the treatment of obesity is justified by the multifactorial pathophysiology of this morbidity, the present project has the following hypotheses: 1. The occurrence of obesity is due to the derange,ent of mitochondrial energy metabolism ; 2. The unbalance is therapeutically modified through physical training ; 3. Obesity courses with the break-down in energy metabolism mitochondrial disease associated with systemic inflammatory characteristics that can be corrected through a combined long-term physical training program. This study have as objective : to analyse changes in mitochondrial function, inflammatory profile, oxidative stress and energy metabolism caused by concurrent physical training in obese women.

NCT ID: NCT03113279 Completed - Physical Activity Clinical Trials

Mechanisms of Age-Related Muscle Loss

Start date: August 1, 2014
Phase: N/A
Study type: Observational

Two independent, but interrelated conditions that have a growing impact on healthy life expectancy and health care costs in developed nations are the age related loss of muscle mass (sarcopenia) and obesity. Sarcopenia affects approximately one third of adults over 60 years of age and more than 50% of those over 80 years, which is of concern when one considers that the most rapidly expanding population demographic in the UK is adults >80 years of age. Skeletal muscle is important in regulating blood glucose and insulin sensitivity. Thus, sarcopenia may play a role in exacerbating insulin resistance and progression toward Type II diabetes (T2D). Indeed, the highest incidence of T2D in the UK has been noted to occur in adults >65 years. Obesity is a major risk factor for chronic diseases including T2D and cardiovascular disease. Progression towards obesity is associated with a concomitant decrease in muscle mass, producing an unfavorable ratio of fat to muscle. Thus, obesity in old age may exacerbate the progression of sarcopenia. For the proposed study the investigators will conduct preliminary laboratory tests to characterize body composition, insulin sensitivity, systemic inflammation, aerobic capacity and muscle protein metabolism (in the fasted and fed state) in healthy older and obese older adults for comparison against healthy young individuals.

NCT ID: NCT03097926 Completed - Clinical trials for Bariatric Surgery Candidate

Duodenal Switch With a Standard Versus Long Alimentary Limb

Long_limb_DS
Start date: May 2013
Phase: N/A
Study type: Interventional

The aim of this study was to determine if Biliopancreatic Diversion with Duodenal Switch (BPD-DS) with a longer strict alimentary limb at the expense of a shorter biliopancreatic limb will offer significant weight lost, but with lesser gastrointestinal complaints, protein deficiency, vitamin and trace element deficit compared to standard BPD-DS.

NCT ID: NCT03082794 Completed - Obesity Clinical Trials

Dietary Effects on Weight Loss and Lipid Profile in Sedentary Men

Start date: February 20, 2011
Phase: N/A
Study type: Interventional

Negative-calorie diet is among the popular dieting guides for weight loss; however, there is still little knowledge about this method. The present study aimed to determine the effects of negative-calorie diet on weight loss and lipid profile, and to compare its efficiency with low-calorie diet in men.

NCT ID: NCT03067428 Completed - Clinical trials for Endothelial Dysfunction

Effects of Fructose Restriction on Liver Steatosis

FRUITLESS
Start date: January 31, 2017
Phase: N/A
Study type: Interventional

Nonalcoholic fatty liver disease (NAFLD) is an emerging health problem as it can lead to end stage liver failure and cardiovascular complications. Diet play an important role in the development of NAFLD. Many studies have addressed the effects of added fructose on NAFLD. To date, little attention has been paid to the effects of a diet devoid of fructose. Therefore, the investigators aim to study the effects of fructose restriction on hepatic fat accumulation and vascular function using a double-blind randomized placebo-controlled design.

NCT ID: NCT03067012 Completed - Gastric Cancer Clinical Trials

Nutritional Safety and Metabolic Benefits of Oncometabolic Surgery for Obese Gastric Cancer Patients

ONCOMETAB
Start date: September 1, 2015
Phase: N/A
Study type: Interventional

The metabolic effect of oncometabolic surgery (long limb Roux-en Y reconstruction) for early gastric cancer patients has been revealed in a few pilot studies. However, the nutritional safety has not been dealt with in previous literatures. This is a prospective pilot study for evaluating the nutritional safety and metabolic benefits of oncometabolic surgery for obese early gastric cancer patients.

NCT ID: NCT03056677 Completed - Clinical trials for Metabolism Disorder, Glucose

Modified Whey Protein and Effect on Post Meal Glucose Levels Study

MWPS
Start date: August 2016
Phase: N/A
Study type: Interventional

After meals, the level of glucose rises in the circulation. In some individuals who are overweight and older, blood glucose can rise to levels which can damage tissues and cause health problems. Usually the hormone insulin, released from the pancreas, effectively lowers blood glucose. However, in overweight and older people insulin is less effective. Certain foods can lower the rise in blood glucose, particularly proteins. This works by increasing the release of a hormone from the gut called Glucagon-Like Peptide 1 (GLP-1), which in turn increases the release of insulin. A Component of milk left over after cheese making, termed Whey protein, is particularly good at releasing GLP-1. Whey protein is used as a food additive and taken as a supplement to help build muscle. Whey protein is a mixture of proteins which the investigators have modified to be more effective at lowering blood glucose. Using laboratory tests the investigators identified a protein present in Whey that does not increase levels of GLP-1 and removed it. It's removal raises the levels of other proteins which are more effective. In this study, the investigators would like to test the effectiveness of the "modified" whey protein. To do this, 30 older, overweight volunteers will be recruited and given the modified whey protein, a normal whey protein or a mixture of amino acids and then a breakfast meal to raise their blood glucose levels. These drinks will be given in a randomised sequence 1 week apart. On each visit, blood samples to measure blood glucose and related hormone levels will be taken. As GLP-1 can also have an effect on appetite, the investigators will measure the effect of the modified whey protein on subsequent appetite in the volunteers by asking them how hungry they feel.