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Right Ventricular Dysfunction clinical trials

View clinical trials related to Right Ventricular Dysfunction.

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NCT ID: NCT03581877 Completed - Clinical trials for Pulmonary Hypertension

Peripheral Systemic Thrombolysis Versus Catheter Directed Thrombolysis for Submassive PE

Start date: January 28, 2019
Phase: Phase 4
Study type: Interventional

To determine whether peripheral low dose systemic thrombolysis (PLST) is non-inferior to catheter directed acoustic pulse thrombolysis (ACDT) in improving RV function and reducing pulmonary artery pressures in submassive pulmonary embolism (PE)

NCT ID: NCT03438825 Completed - Clinical trials for Mitral Valve Disease

PRediction of Early PostoperAtive Right vEntricular Failure in Mitral Valve Replacement/Repair Patients

PREPARE-MVR
Start date: October 2016
Phase:
Study type: Observational

The PREPARE-MVR (PRediction of Early PostoperAtive Right vEntricular failure in Mitral Valve Replacement/Repair patients) Study aims to evaluate those preoperative factors which can predict the early postoperative right ventricular failure or determine the functional shift seen in right ventricular function after mitral valve replacement/repair. The PREPARE-MVR study focuses mainly on echocardiographic (both conventional and advanced) parameters and includes right heart catheterization intraoperatively and in the early postoperative period as gold standard method.

NCT ID: NCT03301571 Completed - Clinical trials for Right Ventricular Dysfunction

Right Ventricular Echocardiography in caRdiac SurgEry

ReVERSE
Start date: May 4, 2017
Phase: N/A
Study type: Interventional

Postoperative right ventricular (RV) dysfunction increases mortality and risk of cardiac failure after cardiac surgery substantially. A comprehensive understanding of this condition is paramount in order to achieve success in treatment and early diagnosis. This study has two main aims. Perioperative aim: To investigate correlations between changes in echocardiographic measurements and hemodynamic changes at baseline and following coronary artery bypass graft (CABG) surgery. Postoperative aim: To evaluate changes in haemodynamics and echocardiographic parameters during separate physiological interventions (increase in preload/afterload, oxygen fraction, pacing modes (AAI/DDD/VVI)).

NCT ID: NCT03183414 Completed - Clinical trials for Right Ventricular Dysfunction

Right Ventricular Dysfunction in Cardiac Surgery Patients

Start date: September 6, 2017
Phase:
Study type: Observational

To establish the correlation between echocardiographic parameters of the RV, measured with TEE and the right ventricular ejection fraction (thermodilution). To identify a time in the perioperative process when RV dysfunction occurs.

NCT ID: NCT01757522 Completed - Clinical trials for Acute Respiratory Distress Syndrome

Detection of Right Ventricular Dysfunction by 2D Strain During Acute Respiratory Distress Syndrom (ARDS)

STRAIN
Start date: January 3, 2013
Phase:
Study type: Observational

Acute respiratory distress syndrome (ARDS) and mechanical ventilation can lead to right ventricular dysfunction and ultimately right ventricular failure by increasing pulmonary vascular resistances and pressure load. This can be prevented by modifying ventilator settings, using vasopressors or inotropes or even by prone positionning.But to do so, right ventricular dysfonction has to be detected. Echocardiography has emerged as a first line tool to diagnose right heart failure. Recently, strain analysis showed promising results to detect early right ventricle abnormalities in other settings such as pulmonary hypertension or scleroderma. We therefore decided to determine whether 2D strain could help detect early right ventricular dysfunction in ARDS.

NCT ID: NCT01571037 Completed - Clinical trials for Right Ventricular Dysfunction

Inhaled Milrinone Use in Patients Receiving HeartMate II LVAD: A Pilot Study

Start date: April 5, 2012
Phase: Phase 1
Study type: Interventional

Right ventricular (RV) failure occurs in an estimated 5-41% of cases involving left ventricular assist device (LVAD) implantation and has been shown to adversely affect peri-operative morbidity and mortality. Current therapies to improve RV dysfunction pre and post-operatively are limited. Inhaled milrinone has been shown in several small human studies to be safely tolerated and provide favorable effects on pulmonary hemodynamics. Study Hypothesis: Delivery of inhaled milrinone, a phosphodiesterase III inhibitor, may provide pulmonary artery vasodilation and therefore improved RV function in patients with end stage heart failure receiving HeartMate II LVAD as a bridge to cardiac transplantation or as destination therapy. Specifically, we aim to: - demonstrate safety of inhaled milrinone in this patient cohort - demonstrate efficacy of inhaled milrinone in this patient cohort

NCT ID: NCT01093001 Completed - Clinical trials for Left Ventricular Dysfunction

Tricuspid Regurgitation Study

Start date: May 2010
Phase: Phase 4
Study type: Interventional

The effect of cardiac pacing leads on tricuspid regurgitation is unclear. This study will determine whether using a smaller diameter leads and an alternate position in the ventricle, the proximal septum, will reduce tricuspid regurgitation than larger leads placed in the apex.

NCT ID: NCT01076296 Completed - Clinical trials for Pulmonary Hypertension

A Post-Marketing Study for On Label Evaluation of the GE Vscan Ultrasound Imaging System

Start date: February 2010
Phase: N/A
Study type: Observational

This study is to collect data and learn more about the Vscan Ultrasound Imaging System. It is an "observational" study with no additional procedures or intervention prescribed other than using the Vscan along with a routine medical physical exam.

NCT ID: NCT00557934 Completed - Clinical trials for Right Ventricular Dysfunction

Right Ventricular Contractility Reserve Following Repair of Tetralogy of Fallot

TOF-Cond
Start date: October 2007
Phase: N/A
Study type: Interventional

Background: Residual pulmonary regurgitation following repair of tetralogy of Fallot, in particular the use of a transannular patch, has been shown to correlate with the development of right ventricular dysfunction. Optimal timing of pulmonary valve replacement, therefore, is important to preserve right ventricular function. Several recent studies suggested that a threshold of right ventricular end-diastolic volume for intervention, in order to preserve the likelihood of adequate reverse remodeling, is in the region of 150 to 200 ml/m2 body surface area. However, there is evidence that right ventricular function does not always recover following pulmonary valve replacement even if the end-diastolic volume is below this cut-off. In addition, previous studies suggested that early dysfunction may be present before symptoms occur. However, early dysfunction is difficult to assess. Methods: Analysis of right ventricular function by pressure-volume loops has been extensively evaluated in experimental studies and is generally considered the optimal way to quantify right ventricular function. By recording a family of pressure-volume loops during reduction of preload, achieved by temporary balloon occlusion of the inferior caval vein, the contractility can be calculated by the slope of the endsystolic pressure-volume relation (elastance). Changes of contractility following dobutamine infusion could be noted by changes of elastance. The increase of the slope during dobutamine demonstrates the contractility reserve of the right ventricle. Purpose: To evaluate the right ventricular contractility reserve to determine early ventricular dysfunction after repair of tetralogy of Fallot.