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Proteinuria clinical trials

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NCT ID: NCT01410994 Completed - Proteinuria Clinical Trials

Bariatric Surgery and the Kidney

Start date: April 2011
Phase: N/A
Study type: Observational

The purpose of the project is to study the relationship between post-bariatric surgery changes in weight and renal parameters.

NCT ID: NCT01386554 Completed - Proteinuria Clinical Trials

Acthar for Treatment of Proteinuria in Membranous Nephropathy Patients

CHART
Start date: August 2011
Phase: Phase 4
Study type: Interventional

The purpose of this study is to provide nephrologists with additional clinical evidence regarding the efficacy and safety of Acthar in subjects with treatment-resistant idiopathic membranous nephropathy. Approximately sixty (60) subjects will be randomized in this double-blind, parallel-group, placebo-controlled, multicenter study comparing Acthar and Placebo administered 2 times per week for a 24-week treatment period followed by a 24-week observation period. The primary objective of this study is to assess the proportion of treatment-resistant subjects (defined as subjects who either have had no response or have suffered a relapse after achieving a partial response to their most recent standard treatment regimen) who have a complete or partial remission of proteinuria in nephrotic syndrome due to idiopathic membranous nephropathy after 24 weeks of treatment.

NCT ID: NCT01382303 Recruiting - Type 2 Diabetes Clinical Trials

Effect of Pentoxifylline on Proteinuria in Korean Type 2 Diabetic Patients

Start date: September 2010
Phase: Phase 4
Study type: Interventional

This multi-center, randomized controlled study aims to evaluate the effects of pentoxifylline on proteinuria in Korean type 2 diabetic patients.

NCT ID: NCT01300273 Active, not recruiting - Clinical trials for Type 2 Diabetic Nephropathy

Low Protein Diet Supplemented With Ketoanalogs on Reducing Proteinuria and Maintaining Nutritional Status in Type 2 Diabetic Nephropathy

Start date: February 2011
Phase: Phase 4
Study type: Interventional

The investigators hypothesize that, LPD supplemented with ketoanalogs will reduce urine podocyte loss and lower the angiotensinogen level in the urine.

NCT ID: NCT01265563 Completed - Oxidative Stress Clinical Trials

N-Acetylcysteine and Milk Thistle for Treatment of Diabetic Nephropathy

CGDN
Start date: January 2011
Phase: Phase 2
Study type: Interventional

The study is done to find out whether the combined use of the nutritional supplements N-acetylcysteine and Siliphos (milk thistle extract) corrects the shedding of urine protein and oxidative damage (damage to cells and organs often compared to fast aging) in patients with Type 2 Diabetes Mellitus (T2DM) and diabetic kidney disease.

NCT ID: NCT01248169 Completed - Clinical trials for Pregnancy; Hypertension, Gestational Hypertension, With Albuminuria (Severe Pre-eclampsia)

Impedance Cardiographic (ICG) Assessment of Pregnant Women With Severe Hypertension to Assess Impact of Standard Therapy

ICASH
Start date: December 2009
Phase: N/A
Study type: Observational

The utilization of external cardiohemodynamic patient assessment, applying non-invasive stick-on contact patches to the mother's neck on either side and chest wall on either side, enables the practitioner to have information about the patient's cardiac function and vascular status beyond simply blood pressure and pulse. This information, once collected, should open the practitioner's eyes to better assess the patient's disease status and her response to therapy. We will use this information to compare the effectiveness of the two standard medications used for treatment of maternal high blood pressure.

NCT ID: NCT01225445 Completed - Clinical trials for Normal Blood Pressure

Treatment of Early Immunoglobulin A Nephropathy by Angiotensin Converting Enzyme Inhibitor - A Randomized Controlled Trial

Start date: April 2002
Phase: Phase 3
Study type: Interventional

Immunoglobulin A (IgA) nephropathy is the most common type of primary glomerulonephritis in the world. The treatment of IgA nephropathy with normal renal function and minimal proteinuria is unknown. Since angiotensin-converting enzyme (ACE) inhibitors reduce proteinuria and retard the rate of decline of renal function in chronic proteinuric nephropathies, including IgA nephropathy. The investigators conduct a randomized control study to evaluate the efficacy of ACE inhibitor in the treatment of early IgA nephropathy. Sixty patients with biopsy-proven IgA nephropathy and minimal proteinuria are recruited. They will be randomized to ramipril for 5 years or no treatment. Blood pressure, proteinuria and renal function will be monitored. This study will explore the effects of ACE inhibitor in the treatment of early IgA nephropathy, which is a major cause of dialysis-dependent renal failure.

NCT ID: NCT01219413 Completed - Clinical trials for Chronic Kidney Disease

Influence of Aliskiren on Proteinuria

ALIPRES
Start date: March 2009
Phase: Phase 4
Study type: Interventional

To evaluate the proteinuria lowering efficacy as well as tolerability and safety of the renin inhibitor aliskiren compared with that of placebo and angiotensin converting enzyme inhibitor perindopril in patients with non-diabetic chronic renal disease.

NCT ID: NCT01169857 Withdrawn - Lupus Nephritis Clinical Trials

Velcade for Proliferative Lupus Nephritis

Start date: August 2010
Phase: Phase 4
Study type: Interventional

The primary objective is to test the safety and efficacy of Velcade to induce remission in WHO class III/IV/V lupus nephritis that are refractory to standard medications.

NCT ID: NCT01164098 Terminated - Proteinuria Clinical Trials

Rituximab to Prevent Recurrence of Proteinuria

Start date: February 2012
Phase: Phase 3
Study type: Interventional

The investigators propose to study novel targets of rituximab in podocytes, with a particular focus on recurrent focal segmental glomerulosclerosis (FSGS). The proposed study has strong clinical implications, since it may extend the approved indications for rituximab treatment to recurrent FSGS as well as to other proteinuric diseases. Furthermore, it will offer new insights into the role of sphyngomyelin related enzymes in podocyte function in health and disease, thus allowing the identification of novel targets for antiproteinuric drug development. Finally, the proposed study offers the opportunity to identify a correlation between the patient's specific clinical outcome and the experimental results obtained after exposing podocytes to patient sera in the presence or absence of rituximab. Therefore, it may lead to the development of an assay for the pre-transplant identification of patients at high-risk for recurrent disease and, among them, may allow the identification of those patients that will respond to rituximab.