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Obstructive Sleep Apnoea clinical trials

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NCT ID: NCT02518633 Completed - Obesity Clinical Trials

Obstructive Sleep Apnoea and Adipose Tissue Dysfunction

Start date: March 2010
Phase: N/A
Study type: Observational

Dysfunctional adipose tissue predisposes to cardiovascular disease. Similarly, the risk of cardiovascular disease appears to be increased in subjects with obstructive sleep apnoea. Reduced adipose tissue oxygen availability has been described in obesity and may also be a mechanism in obstructive sleep apnoea. Hypoxia induces inflammation and fibrosis in adipose tissue which are factors contributing to cardiovascular risk. The investigators hypothesize that adipose tissue's oxygen uptake is reduced in subjects with obstructive sleep apnoea by comparing in vivo AT oxygenation and blood flow in tissue of control subjects.

NCT ID: NCT02417584 Completed - Clinical trials for Obstructive Sleep Apnoea

Efficacy of PAP Therapy on Blood Pressure

Start date: June 2009
Phase: N/A
Study type: Interventional

The aim of the study was to determine the long term effects of compliance with positive airway pressure therapy on blood pressure in both hypertensive and normotensive patients with obstructive sleep apnoea hypopnoea syndrome.

NCT ID: NCT02015598 Completed - Clinical trials for Obstructive Sleep Apnoea

Antioxidant Carbocysteine Treatment in Obstructive Sleep Apnoea Syndrome

Start date: December 2013
Phase: N/A
Study type: Interventional

Obstructive sleep apnoea syndrome (OSAS) is characterised by repeated episodes of upper airway occlusion during sleep.It can cause cycles of hypoxia reoxygenation. And it was postulated that intermittent hypoxia seems to resemble ischemia-reperfusion.Many study suggest that ischemia-reperfusion represents an oxidative stress causing increased generation of reactive oxygen species, especially superoxide anions.It is one of the most important mechanisms of cardiovascular diseases, including hypertension, coronary artery disease and cerebrovascular accident complication with OSAS.So many individuals approve OSAS is an Oxidative Stress disease. Continuous positive airway pressure (CPAP) is the first-line of treatment method in moderate/severe OSA.But poor adherence to CPAP treatment is very common.The failure rate with CPAP treatment is more than 50%.So we are searching a new treatment for that patients. Carbocysteine is a antioxidant.It can not only scavenges the free radicals but also replenishes glutathione(GSH)which is has double antioxidant capacity. However, Carbocysteine is cheaper than other which has double antioxidant capacity drugs,such as N-acetylcysteine.The purpose is to evaluate efficacy of oral intake of Antioxidant Carbocysteine witch can reduce oxidative stress and improve the symptom of OSAS.It recover the imbalance in the oxidant-anti-oxidant status may reduce cardiovascular abnormalities in Patients with OSAS.

NCT ID: NCT01797653 Completed - Clinical trials for Obstructive Sleep Apnoea

Randomized-controlled Trial (RCT) on CPAP-withdrawal and Microvascular Function in OSA

Start date: February 2013
Phase: N/A
Study type: Interventional

Randomized controlled trial (therapeutic vs. placebo CPAP) including 46 patients with obstructive sleep apnea (OSA) to define the physiological effects of continuous positive airway pressure (CPAP) therapy withdrawal on myocardial perfusion and dermal and renal microvascular function during a 2 week period.

NCT ID: NCT01687855 Completed - Clinical trials for Obstructive Sleep Apnoea

Dynamic Alterations of Tongue in Sleep Apnoea Hypopnea Syndrome During Sleep

Start date: February 2003
Phase: N/A
Study type: Observational

The aim of this study was to evaluate the dynamic changes of the tongue in patients with obstructive sleep apnoea hypopnea syndrome (OSAHS) during sleep and while awake by using Ultrafast Magnetic Resonance Imaging (UMRI)

NCT ID: NCT01661712 Completed - Clinical trials for Obstructive Sleep Apnoea

Continuous Transcutaneous Electrical Stimulation in Sleep Apnoea

TESLA
Start date: March 2013
Phase: N/A
Study type: Interventional

The aim of this randomized, double-blinded, sham-controlled cross-over trial is to demonstrate the effectiveness of continuous transcutaneous electrical stimulation of the pharyngeal dilator muscles to reduce sleep-disordered breathing.

NCT ID: NCT01620554 Completed - Clinical trials for Obstructive Sleep Apnoea

Dose-range Finding Study of BF2.649 Effect on Patients With Obstructive Sleep Apnea (OSA)

Start date: October 2010
Phase: Phase 2
Study type: Interventional

Prospective, multicenter, double-blind, phase II, randomized, dose-response study in 5 parallel groups (dose-range).

NCT ID: NCT01619748 Completed - Obesity Clinical Trials

Peripheral Vascular Function in Obstructive Sleep Apnoea

Start date: June 2012
Phase: N/A
Study type: Observational

Peripheral vascular function is impaired in people with obstructive sleep apnoea who are untreated compared with age-, weight- and sex-matched healthy controls.

NCT ID: NCT01557166 Completed - Obesity Clinical Trials

Effect of Liraglutide in Obese Subjects With Moderate or Severe Obstructive Sleep Apnoea: SCALEā„¢ - Sleep Apnoea

Start date: June 7, 2012
Phase: Phase 3
Study type: Interventional

This trial is conducted in North America. The aim of the trial is to investigate the effect of liraglutide in obese subjects with sleep apnoea.

NCT ID: NCT01546792 Completed - Obesity Clinical Trials

Lifestyle Intervention in Obstructive Sleep Apnoea

Start date: January 2010
Phase: Phase 2/Phase 3
Study type: Interventional

Obstructive sleep apnoea/hypopnoea syndrome (OSAHS) is a common disorder characterised by interrupted breathing while sleeping. It is associated with cardiovascular problems such as high blood pressure, heart attack and stroke. Furthermore, most patients with OSAHS are overweight and have impaired quality of life. Lifestyle interventions incorporating exercise training, dietary advice and behaviour change have been shown to elicit favourable changes in quality of life, body mass/composition and cardiovascular risk in a range of patient groups. However, no study has investigated the impact of lifestyle modification on such health outcomes in patients with OSAHS. This study will address this issue by investigating the effects of a lifestyle intervention on quality of life, body mass/composition and cardiovascular risk in patients with OSAHS. A total of 60 volunteers will be recruited and randomly allocated to one of the two groups. Patients in the intervention group will be offered a 12-week individualised lifestyle programme consisting of supervised exercise training, dietary advice and behaviour change counselling. Patients in the control group will receive an educational booklet detailing healthy eating and exercise guidelines but no supervised or structured intervention. The results of this study will inform the design of a larger, multi-centre randomised controlled trial.