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Obesity, Adolescent clinical trials

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NCT ID: NCT06213922 Not yet recruiting - Obesity, Adolescent Clinical Trials

Prevalence of and Risk Factors of Obesity in School Children and Adolescents in Nag Hammadi City. 300 Participants.

Start date: January 2024
Phase:
Study type: Observational [Patient Registry]

Searching for obese children who's body mass index is above normal range and risk factors in children and adolescents in Nag Hammadi city

NCT ID: NCT05984043 Not yet recruiting - Obesity, Adolescent Clinical Trials

Piloting a Biofeedback Intervention for Adolescents With Overweight and a History of Adverse Childhood Experiences

Start date: November 2023
Phase: N/A
Study type: Interventional

Adverse childhood experiences (ACEs), referring to traumatic experiences occurring before 18 years of age (e.g., abuse/neglect), are associated with a 30-70% increased risk of developing adolescent and adult obesity and obesity-related cardiometabolic comorbidities, which may be due to dysregulation in stress-related physiology and engagement in stress-related behaviors. While adolescents with ACEs have a 2.25 times increased risk of obesity, standard-of-care lifestyle (e.g., nutrition/physical activity) interventions do not typically incorporate trauma-informed care, including assessment and therapeutic attention to effects of ACEs. Heart rate variability (HRV) biofeedback is an evidence-based, relatively brief, mind-body intervention targeting the stress physiology that can be dysregulated in adolescents with ACEs. The objective of this proposal is to adapt and test a 4-session HRV biofeedback protocol for 12-17-year-olds with BMI>85th percentile and ACEs. First (Phase 1), we will iteratively adapt a 4-session HRV biofeedback facilitator protocol with n=3-5 adolescents with overweight/obesity and ACEs, using adolescent quantitative and qualitative feedback to hone and optimize HRV biofeedback for this population (Aim 1). Next (Phase 2), we will conduct a randomized waitlist-controlled pilot study of n=30 adolescents with overweight/obesity and ACEs to assess acceptability and feasibility (Aim 2) and to describe changes in theorized targets of biofeedback (Aim 3).

NCT ID: NCT05424094 Not yet recruiting - Clinical trials for Overweight and Obesity

HRQoL in Overweight and Obese Children/Adolescents

Start date: July 1, 2022
Phase: N/A
Study type: Interventional

The primary aim of the present study is to record changes in the health-related quality of life (HRQoL) of children/adolescents participating in the multi-professional group programme 'kinderleicht' in 2022 over the course of the eleven-month active phase. In addition, these data will be examined for correlation with changes in BMI, waist and hip circumference, and motor performance. Secondarily, the study addresses the questions of whether the recording of HROoL by means of the KINDL questionnaire is practicable for the programme leaders and what benefit can be gained from the analysed data for future cohorts.

NCT ID: NCT04100109 Not yet recruiting - Obesity, Childhood Clinical Trials

Evaluating the Metabolic Effects of Polylactose: A Novel Prebiotic

Start date: August 1, 2024
Phase: N/A
Study type: Interventional

This study will evaluate the feasibility and obtain a preliminary estimate of efficacy of feeding 15 g/day of a new novel prebiotic dietary fiber, termed polylactose, in 40 children (8-12 years old) with obesity (body mass index [BMI]-percentile >/= 95th) who have magnetic resonance imaging (MRI)-confirmed non-alcoholic fatty liver disease [NAFLD] (hepatic fat fraction >/= 5.5%), compared to a placebo of 15 g/d of cellulose, an inert dietary fiber.

NCT ID: NCT02973867 Not yet recruiting - Obesity, Adolescent Clinical Trials

Myokines Role During Obesity

Bonamy
Start date: November 2016
Phase: N/A
Study type: Observational

We showed, for the first time, that Z-score increase during obesity was dependent on sex, severity of obesity and age but also on the bone site. Although the increase in BMD at bearing areas seems to be logically explained by the assumption that mechanical stresses have an osteogenic effect on bone tissue, BMD can in no way respond to the concomitant increase in BMD at non-load bearing areas such as the radius. These results suggest that changes in certain systemic compounds, induced by obesity, may interfere with bone metabolism. Moreover, the fact that the BMD or Z score is positively and only correlated with muscle mass and not with fat mass confirm that mechanical stresses are not the only factors acting on bone tissue. Our objectives are to determine the relationship between plasma concentration of myokines, adipokines, bone markers and BMD in obese subjects.