Clinical Trials Logo

Bone Loss clinical trials

View clinical trials related to Bone Loss.

Filter by:

NCT ID: NCT01228318 Completed - Osteoporosis Clinical Trials

Bone Loss and Immune Reconstitution in HIV/AIDS (BLIR-HIV)

BLIR-HIV
Start date: January 2011
Phase: Phase 2
Study type: Interventional

With the increasing age of people living with HIV/AIDS, age-induced osteoporosis is likely to be compounded by HIV/AIDS and HAART-associated bone loss. Mechanistically, osteoclasts the cells responsible for bone resorption form under the influence of the key osteoclastogenic cytokine receptor activator of nuclear factor kappa-Β ligand (RANKL). The osteoclastogenic and proresorptive activities of RANKL are moderated by its physiological decoy receptor osteoprotegerin (OPG). Imbalance in the ratio of RANKL to OPG alters osteoclastic bone resorption and lead to osteoporosis. Activated T- and B-cells are a major source of RANKL, while normal physiological B-cells are a major source of OPG. T-cells regulate the production of OPG by B-cells. Thus changes in the immune system induced by HIV/AIDS and/or by HAART could affect B-cell and T-cells RANKL and OPG production. Indeed, data from our group shows that in an animal model of HIV/AIDS, the HIV-1 Transgenic rat, the development of osteoporosis is recapitulated as observed in HIV-infected patients, and B-cell OPG and RANKL production are concurrently down regulated and upregulated respectively. Furthermore, preliminary data in HIV-infected subjects suggests dramatic acute upswing in bone resorption following HAART initiation that peaks at 12 weeks and then declines. Based on these findings, the investigators hypothesize HAART associated bone loss is driven by immune reconstitution. Because this effect of HAART is dramatic in magnitude but short in duration, the investigators propose to apply antiresorptive agent (zoledronic acid, reclast®) to specifically spare patients from this dramatic but acute bone damage.

NCT ID: NCT01225055 Completed - Osteoporosis Clinical Trials

Effect of Teriparatide, Vibration and the Combination on Bone Mass and Bone Architecture in Chronic Spinal Cord Injury

Start date: October 2010
Phase: Phase 2
Study type: Interventional

Spinal cord injury (SCI) results in marked acute loss of bone. This study evaluates the effect of teriparatide (PTH) and the use of vibration as a form of mechanical stimulation on bone mass.

NCT ID: NCT01102985 Completed - Cancer Clinical Trials

Women With Cancer: An Exercise Study to Promote Health

Start date: January 2008
Phase: N/A
Study type: Interventional

The purpose of the study is to evaluate the effect of an aerobic-resistance exercise program compared to a home based physical activity program on bone mass, body composition, metabolic risk factors and cardiovascular fitness in women with cancer who have completed therapy.

NCT ID: NCT00975013 Completed - Bariatric Surgery Clinical Trials

Bone Loss After Laparoscopic Roux-en-Y Gastric Bypass

Start date: April 2007
Phase: N/A
Study type: Observational

The objective of this study is to assess changes bone mineral density and bone metabolism after laparoscopic Roux-en-Y gastric bypass surgery. The investigators hypothesize that weight loss after laparoscopic Roux-en-Y gastric bypass surgery will be associated with increased bone turnover, changes in bone metabolism, and loss of bone mass.

NCT ID: NCT00901121 Completed - Bone Loss Clinical Trials

Maxillary Sinus Elevation With Bio-Oss or Straumann BoneCeramic

Start date: July 2005
Phase: N/A
Study type: Interventional

The purpose of the study was to test a clinically relevant non-inferiority of Straumann BoneCeramic treatment compared to Bio-Oss treatment 180 to 240 days after sinus grafting procedure.

NCT ID: NCT00900718 Completed - Bone Loss Clinical Trials

Comparison of Straumann Bone Ceramic and Bio-Oss With Guided Tissue Regeneration for Alveolar Ridge Preservation

Start date: February 2006
Phase: N/A
Study type: Interventional

The purpose of this study is to compare a new biomaterial with an already existing treatment principle (Bio-Oss/Bio-Gide) with regard to their ability to preserve the dimensions of the alveolar ridge and to promote bone regeneration and to evaluate the early survival rate of dental implants placed in the augmented socket area and to assess radiographically and clinically changes of peri-implant soft and hard tissues.

NCT ID: NCT00869622 Completed - Epilepsy Clinical Trials

Antiepileptic Drugs and Osteoporotic Prevention Trial

ADOPT
Start date: June 2006
Phase: Phase 4
Study type: Interventional

Study Design: (e.g., Controlled, Double-Blind, Randomized, Parallel): Randomized, double-blind, placebo controlled of a bisphosphonate in the prevention of bone loss associated with the use of antiepileptic drugs.

NCT ID: NCT00826228 Completed - Osteoporosis Clinical Trials

Effect of Parathyroid Hormone (PTH) and Weight-Bearing on Bone in Spinal Cord Injury (SCI)

Start date: June 2009
Phase: Phase 4
Study type: Interventional

Individuals with spinal cord injury sustain significant loss of bone mass in their lower extremities (20-40% or more). This study evaluates the ability of PTH and weight-bearing, two interventions that build bone, to increase bone mass in this population.

NCT ID: NCT00772395 Completed - Bone Loss Clinical Trials

Can Risedronate Prevent Periprosthetic Bone Loss After Total Hip Arthroplasty

PREVENT
Start date: April 2006
Phase: Phase 4
Study type: Interventional

Can risedronate given once weekly for 6 months prevent periprosthetic bone loss after uncemented total hip arthroplasty?

NCT ID: NCT00738257 Completed - Bone Loss Clinical Trials

A Prospective, Multicentre, Open-label Randomised Controlled Study to Investigate the Effectiveness of Pamidronate in the Prevention of Bone Loss in de Novo Renal Transplant Patients.

SMART
Start date: June 2000
Phase: Phase 4
Study type: Interventional

The purpose of the study was to evaluate the effect of Pamidronate on bone mineral density loss and fracture rates up to 2 years, in post renal-transplant subjects on a Ciclosporin A and glucocorticoid based immunosuppressive regimen.