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Clinical Trial Summary

Amyloid plaques and tau protein are the landmarks of neurodegeneration in Alzheimer's disease (AD). On the other hand, it is reported that cerebral ischemia may induce amyloid plaques and tau protein accumulation. However, it was difficult to in vivo disentangle the complex and dynamic interactions between AD pathophysiology and cerebral vascular injury in the development of post-stroke cognitive impairment in the past. With the advent of novel radiotracers specific to cerebral amyloid plaques and tau protein, we aim to conduct a prospective multimodal neuroimaging cohort study to investigate the contribution of vascular injury, amyloid plaques and tau protein to stroke recovery and post-stroke cognitive impairment.


Clinical Trial Description

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Study Design


Related Conditions & MeSH terms


NCT number NCT04572477
Study type Interventional
Source Chang Gung Memorial Hospital
Contact Huang Kuo-Lun, M.D.
Phone +886-3-3281200
Email drkuolun@cgmh.org.tw
Status Recruiting
Phase Phase 2
Start date June 1, 2017
Completion date May 31, 2024