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Hyperplasia clinical trials

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NCT ID: NCT00651807 Terminated - Clinical trials for Benign Prostatic Hyperplasia (BPH)

A Trial to Investigate the Effectiveness and Safety of Org 3236 (Etonogestrel) Tablets in Men With Urinary Complaints Suggestive of a Benign Enlargement of the Prostate (304001)(P05806)

Start date: March 2008
Phase: Phase 2
Study type: Interventional

This trial is conducted to evaluate the effect of etonogestrel in comparison to placebo on: - the prostate volume and the urinary complaints; - the urinary flow and the urinary volume in the bladder after voiding; - the progression of the disease; - the sexual function, well-being and urinary complaints-related Quality of Life. In addition the safety and the way the drug is absorbed and excreted by the body will be analyzed.

NCT ID: NCT00648323 Completed - Prostate Clinical Trials

Evaluation Of The Efficacy And Safety Of The Doxasozin Gastrointestinal Therapeutic System (GITS) In Patients With Prostate Enlargement

Start date: November 2003
Phase: Phase 4
Study type: Interventional

The primary objectives were to determine the efficacy and safety of the GITS formulation of Doxazosin in Taiwanese patients with prostate enlargement.

NCT ID: NCT00645034 Completed - Clinical trials for Benign Prostatic Hyperplasia

A Study to Investigate the Effects on Blood Pressure and Pulse Rate of a Single 100mg Dose of Sildenafil in Patients With Benign Prostatic Hyperplasia Who Are Being Treated With Doxazosin

Start date: November 2002
Phase: Phase 2
Study type: Interventional

To investigate the effects of a single dose of sildenafil (100 mg) or placebo on blood pressure and pulse rate in subjects taking doxazosin for benign prostatic hyperplasia (BPH). To investigate the pharmacokinetics of doxazosin when co-administered with sildenafil 100 mg, and to investigate the safety and toleration of sildenafil 100 mg when co-administered with doxazosin.

NCT ID: NCT00637715 Completed - Clinical trials for Benign Prostatic Hyperplasia

Once Daily Given Alfuzosin in the Treatment of BPH

Start date: October 2003
Phase: Phase 4
Study type: Interventional

Collection of the data on the safety and efficacy of the once daily administration of the alfuzosin preparation /Alfetim Uno® l0 mg/ at patients with lower urinary tract symptoms/complaints rendering possible the presence of benign prostatic hyperplasia, in the course of everyday practice

NCT ID: NCT00637481 Completed - Breast Cancer Clinical Trials

A Phase I Prevention Study of Atorvastatin in Women at Increased Risk for Breast Cancer

Start date: March 2008
Phase: Phase 1
Study type: Interventional

Chemoprevention is the use of certain drugs to keep cancer from forming. The use of atorvastatin (Lipitor) may prevent breast cancer. This randomized phase I trial is studying the best dose of atorvastatin in preventing breast cancer in women at increased risk for breast cancer.

NCT ID: NCT00627276 Completed - Breast Cancer Clinical Trials

Omega-3 Fatty Acids in Treating Women With Newly Diagnosed Ductal Carcinoma In Situ and/or Atypical Ductal Hyperplasia

Start date: December 2007
Phase: N/A
Study type: Interventional

RATIONALE: Polyunsaturated fatty acids are important for normal growth and development. One type, called omega-3 fatty acids (found in fish, fish oil, and some other foods), may affect the growth of abnormal breast cells. PURPOSE: This randomized pilot trial is studying how well omega-3 fatty acids work in treating women with newly diagnosed ductal carcinoma in situ and/or atypical ductal hyperplasia.

NCT ID: NCT00623740 Completed - Clinical trials for Bronchopulmonary Dysplasia

PREMILOC Trial to Prevent Bronchopulmonary Dysplasia in Very Preterm Neonates

PREMILOC
Start date: April 2008
Phase: Phase 3
Study type: Interventional

There is increasing evidence linking a fetal and early neonatal systemic inflammatory response syndrome to the subsequent development of bronchopulmonary dysplasia (BPD) and white matter injury (WMI) in very preterm infants. Babies with evidence of adrenal insufficiency early in life may not be able to control the inflammatory response and are thereby more likely to develop BPD than babies who do not show such evidence of inflammation. We designed a randomized controlled trial to test the hypothesis whether very preterm babies at high-risk of BPD, treated with low doses of HC during the first 10 days of life, are more likely to survive without BPD at 36 weeks of post-menstrual age (PMA), compared to babies treated with placebo.

NCT ID: NCT00621985 Completed - Clinical trials for Adrenal Hyperplasia, Congenital

Dexamethasone Treatment of Congenital Adrenal Hyperplasia

Start date: April 2008
Phase: Phase 2
Study type: Interventional

The purpose of this study is to determine if dexamethasone given at night is a more effective treatment for congenital adrenal hyperplasia in young children than standard three times per day hydrocortisone. Our hypothesis is that nocturnal dexamethasone will lead to more efficient suppression of the hypothalamic-pituitary-adrenal axis. We performed a cross-over trial comparing hormonal control during two 24-hour hospitalizations, one on hydrocortisone and one on nocturnal dexamethasone.

NCT ID: NCT00620087 Completed - Clinical trials for Atypical Ductal Hyperplasia

Molecular Breast Imaging in Women With Atypia and LCIS

Start date: August 2003
Phase: N/A
Study type: Interventional

We aim to determine if Molecular Breast Imaging (a new nuclear medicine technique developed at Mayo) can identify malignant breast lesions in women who have atypical ductal hyperplasia, atypical lobular hyperplasia, or lobular carcinoma in situ.

NCT ID: NCT00617292 Recruiting - Clinical trials for Adrenal Hyperplasia, Congenital

Determining the Long-Term Effects of Prenatal Dexamethasone Treatment in Children With 21-Hydroxylase Deficiency and Their Mothers

Start date: January 2008
Phase: N/A
Study type: Observational

Congenital adrenal hyperplasia (CAH) is a genetic disorder that affects the amount of steroids that the body forms. The most common form of CAH is 21-hydroxylase deficiency (21OHD), which leads to cortisol deficiency and causes the development of mature masculine characteristics in newborn, prepubescent, and grown females, and prepubescent males. Prenatal treatment with dexamethasone, a corticosteroid, has been shown to reduce the masculinization of genitalia. However, the long-term effects of dexamethasone on the children who received it as fetuses and on mothers who were exposed to it while they were pregnant have not been determined. This study will investigate potential long-term adverse side effects of prenatal dexamethasone treatment in children and young adults who received dexamethasone as fetuses and their mothers who were exposed to it during pregnancy.