Chronic Periodontitis Clinical Trial
The etiopathogenesis of periodontal disease results from complex interaction between infectious agents, mainly including bacteria, and host cellular and humoral immune responses. However it is thought that bacteria-induced pathogenesis is not sufficient alone to explain all biological and clinical features of the destructive periodontal disease. The main hypothesis is that herpesviruses, such as Epstein-Barr Virus, may participate as well by altering epithelial gingival cell biology and consequently may promote the initiation and progression of periodontitis.
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Status | Clinical Trial | Phase | |
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Not yet recruiting |
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Completed |
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N/A | |
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Phase 1/Phase 2 | |
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Phase 2 | |
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Phase 2/Phase 3 | |
Terminated |
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N/A | |
Completed |
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Leptin and Visfatin in Diabetic Patients With Periodontitis Before and After Periodontal Therapy
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N/A | |
Completed |
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Benefits of Platelet Rich Fibrin In Mandibular Molar Furcation Defects
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N/A | |
Completed |
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N/A | |
Completed |
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N/A | |
Completed |
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Treatment of Intrabony Periodontal Defects With Enamel Matrix Derivatives and Autogenous Bone Graft
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Phase 4 | |
Completed |
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Antimicrobial Therapy as Adjunct to Periodontal Treatment: Effect of Timing
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Phase 4 | |
Not yet recruiting |
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Phase 2 | |
Not yet recruiting |
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Completed |
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The Adjunctive Effect of Probiotics to Non Surgical Treatment of Chronic Periodontitis
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Phase 1 | |
Completed |
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N/A | |
Completed |
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N/A | |
Completed |
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Phase 2/Phase 3 | |
Completed |
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N/A | |
Completed |
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N/A |