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Peripheral Vascular Diseases clinical trials

View clinical trials related to Peripheral Vascular Diseases.

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NCT ID: NCT03238222 Recruiting - Clinical trials for Peripheral Arterial Disease

Motivating Structured Walking Activity in Intermittent Claudication

MOSAIC
Start date: November 10, 2017
Phase: N/A
Study type: Interventional

Peripheral arterial disease can cause leg pain or discomfort (called intermittent claudication (IC)), which limits the ability to walk and carry out everyday activities. Lifestyle changes, like increasing walking, can lead to improvements, but can be a challenge to begin and then continue. The aim of this study is to investigate if a physiotherapist-led, behaviour change treatment effects walking in people with IC. The treatment is designed to build an understanding of IC and walking exercise and help individuals develop strategies to increase regular walking. The trial will recruit 192 adults (aged 50 years and over) with IC. All participants will visit King's College London or their local hospital to complete two short walking tests and answer questionnaires about their daily activities, beliefs about their symptoms and treatment, quality of life and the costs of having IC. Participants will then be randomly assigned to receive either the new treatment or continue with their usual NHS care. The new treatment involves two 60-minute face-to-face sessions (at the participant's home or local hospital) and two 20-minute telephone calls with a physiotherapist who will discuss participants' understanding and beliefs about IC, walking and help participants to set goals and plans to increase walking over 12 weeks. Participants will be provided with a step counter (pedometer) and walking record. After 12 weeks, all participants will attend a second appointment where they will repeat the walking tests and fill out another set of questionnaires. A final set of questionnaires will be completed by all participants after 6 months (by post or electronically). Some participants will be invited to provide feedback on their experience of the treatment and trial by telephone or a face-to-face interview with a researcher.

NCT ID: NCT03195556 Recruiting - Clinical trials for Peripheral Arterial Disease

Augmentation of Limb Perfusion With Contrast Ultrasound

Start date: June 18, 2018
Phase: N/A
Study type: Interventional

Our laboratory has discovered that ultrasound (US) imaging together with clinically approved microbubble ultrasound contrast agents can augment limb tissue perfusion. These observations have been made in mice with and without peripheral artery disease (PAD), and also in humans where high power contrast enhanced ultrasound (CEU) was used to measure perfusion but was found also to augment perfusion by almost 2-fold. The latter human studies were performed with ultrasound protocols designed for perfusion imaging and not for flow augmentation. In this study, we will measure the degree to which limb perfusion is augmented with specific therapeutic CEU settings that are still within the FDA-approved limits with regards to US power and contrast dosing.

NCT ID: NCT03121430 Recruiting - Clinical trials for Peripheral Arterial Disease

Study of Drug Eluting Peripheral Vascular Stent System in Superficial Femoral Artery Stenosis and /or Occlusion

Start date: October 22, 2018
Phase: N/A
Study type: Interventional

Evaluation of drug eluting peripheral vascular stent system for the treatment of superficial femoral artery stenosis and / or occlusion, there is better in effectiveness and clinical performance compared with similar products already on the market.

NCT ID: NCT03092336 Recruiting - Clinical trials for Peripheral Arterial Disease

Effects of Different Protocols of Physical Training on Levels of Muscle Strength and Functional Capacity

Start date: May 2011
Phase: N/A
Study type: Interventional

Introduction: the peripheral obstructive arterial disease is an condition that affects around 15% of the world population interfering in muscle strength, life quality and functional capacity of patients.

NCT ID: NCT03058913 Recruiting - Clinical trials for Peripheral Arterial Disease

OEIS Outcome Data Registry

Start date: February 1, 2017
Phase:
Study type: Observational [Patient Registry]

This registry is created under the sponsorship of the Outpatient Endovascular and Interventional Society (OEIS) to monitor the clinical course and treatment outcomes of patients undergoing office-based endovascular procedures.

NCT ID: NCT02965677 Recruiting - Clinical trials for Peripheral Arterial Disease

A Study of The Femoral Popliteal Artery Treated With LEGFLOW OTW

Start date: December 2016
Phase: N/A
Study type: Interventional

To evaluate the safety and efficacy of the Paclitaxel Releasing Peripheral Balloon Dilatation Catheter (LEGFLOW) compared with the standard balloon (Admiral Xtreme) for the treatment of stenosis or occlusions in femoral popliteal artery.

NCT ID: NCT02959606 Recruiting - Clinical trials for Peripheral Arterial Disease

SAFE (Sarpogrelate Anplone in Femoro-popliteal Artery Intervention Efficacy) Study

Start date: December 2016
Phase: Phase 4
Study type: Interventional

After endovascular treatment (EVT) for peripheral artery disease (PAD), dual antiplatelet therapy (DAAT) of aspirin (ASA) and clopidogrel are currently drug of choice to prevent occlusion. Anplone SR®, controlled-released Sarpogrelate hydrochloride, has been introduced as an anti-platelet agent for the drug of PAD. The aim of this study was to compare the efficacy and safety of Anplone + aspirin and clopidogrel + aspirin in patients who underwent EVT for femoro-popliteal occlusive disease.

NCT ID: NCT02915796 Recruiting - Clinical trials for Peripheral Arterial Disease

Autologous CD133(+) Cells as an Adjuvant to Below the Knee Percutaneous Transluminal Angioplasty

Start date: September 2016
Phase: Phase 1
Study type: Interventional

The main aim of the present study was to evaluate the therapeutic potential and safety of transarterial infusion of granulocyte colony stimulating factor (G-CSF) mobilized cluster of differentiation (CD) 133(+) cells when combined with percutaneous transluminal angioplasty (PTA) in treatment of below the knee (BTK) peripheral arterial disease (PAD) in diabetic patients.

NCT ID: NCT02850432 Recruiting - Clinical trials for Peripheral Arterial Disease

Clinical and MRI Cardiac Biomarkers on Peripheral Arterial Disease in Patients With Long Term DM

CMRPADDM
Start date: May 1, 2016
Phase: N/A
Study type: Interventional

The aim of this study is to establish a clinically feasible simultaneous evaluation of heart and lower limb using MRI and to assess the combined benefit of clinical and cardiac MRI imaging markers in the evaluation of PAD and prediction of treatment outcome.

NCT ID: NCT02842424 Recruiting - Clinical trials for Peripheral Arterial Disease

Ramipril Treatment of Claudication: Oxidative Damage and Muscle Fibrosis

Start date: February 25, 2016
Phase: Phase 4
Study type: Interventional

Peripheral artery disease (PAD) is a manifestation of atherosclerosis that produces progressive narrowing and occlusion of the arteries supplying the lower extremities. The most common clinical manifestation of PAD is claudication, i.e., a severe functional limitation identified as gait dysfunction and walking-induced leg muscle pain relieved by rest. The standard therapies for claudication include the medications cilostazol and pentoxifylline, supervised exercise therapy and operative revascularization. Recent data demonstrated that 24 weeks of treatment with the angiotensin-converting enzyme (ACE) inhibitor Ramipril produces improvements in the walking performance of patients with claudication that are higher than those of cilostazol and pentoxifylline and similar to those produced by supervised exercise therapy and operative revascularization. The mechanisms by which Ramipril therapy produces this impressive improvement in the functional capacity of claudicating patients remain unknown. The Investigators hypothesize that treatment of claudicating PAD patients with Ramipril will improve walking performance and quality of life by improving the myopathy of the gastrocnemius. Improved myopathy is a consequence of reduced oxidative damage, reduced TGF-β1 production by vascular smooth muscle cells and reduced collagen deposition in the affected gastrocnemius.