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Pelvic Pain clinical trials

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NCT ID: NCT00826514 Completed - Clinical trials for Chronic Prostatitis With Chronic Pelvic Pain Syndrome

An Efficacy And Safety Study Of Tanezumab For The Treatment Of Pain Associated With Chronic Abacterial Prostatitis

Start date: March 25, 2009
Phase: Phase 2
Study type: Interventional

The purpose of this study is to determine whether tanezumab is effective in the treatment of pain associated with chronic prostatitis.

NCT ID: NCT00817388 Completed - Pelvic Pain Clinical Trials

Correlating Ic/Pbs Symptoms With Urine Biomarkers

Start date: January 2009
Phase: N/A
Study type: Observational

Completion of a brief survey tool and providing an urine specimen. The study purpose is to improve our understanding of interstitial cystitis and painful pelvic syndrome.

NCT ID: NCT00778609 Completed - Headache Clinical Trials

Effect of a New Oral Contraceptive Pill on Hormone Related Symptoms Such as Pelvic Pain and Headache

Start date: December 2008
Phase: Phase 3
Study type: Interventional

The aim of the present study is to investigate whether women taking a new combined oral contraceptive pill (SH T00658ID, estradiol valerate/dienogest) experience fewer hormone withdrawal-associated symptoms such as pelvic pain or headache during their monthly cycle compared to a commonly used contraceptive pill (Microgynon).

NCT ID: NCT00775281 Completed - Overactive Bladder Clinical Trials

Changes in Inflammatory and Contractile Protein Expression in Patients With Painful Bladder Syndrome/IC.

Start date: October 2006
Phase: N/A
Study type: Observational

Interstitial cystitis (IC)/chronic pelvic pain syndrome (CPPS) is a clinical syndrome of pelvic pain and/or urinary urgency/frequency in absence of a specific cause such as bacterial infection or damage to the bladder. The pathogenetic mechanisms of IC/CPPS are as yet undefined and it is largely this lack of knowledge, which precludes a systematic therapeutic approach. Experimental evidence, including results from the animal models of cystitis and the knock-out mice, indicate a participation of tachykinin receptors, especially the NK1R, in neurogenic inflammation, which is considered an important element of the IC complex. However, there is very scant information about the molecular mechanisms of IC in humans, or of the types of receptors, which participate in neurogenic inflammation. Based on our molecular biological know-how and the clinical expertise, we propose to investigate the role of the tachykinin and bradykinin receptors and their signalling partners in CPPS and bladder dysfunction in humans.

NCT ID: NCT00740779 Completed - Clinical trials for Abacterial Chronic Prostatitis/Chronic Pelvic Pain Syndrome

Use of Silodosin to Treat Moderate to Severe Abacterial Chronic Prostatitis/Chronic Pelvic Pain Syndrome.

Start date: September 2008
Phase: Phase 2
Study type: Interventional

The primary objective is to compare the efficacy of silodosin 4 and 8 mg once daily with placebo in the treatment of subjects with moderate to severe abacterial chronic prostatitis/chronic pelvic pain syndrome during a 12 week treatment period. The secondary objective is to compare the safety of silodosin 4 and 8 mg once daily with placebo.

NCT ID: NCT00735852 Completed - Endometriosis Clinical Trials

Decapeptyl SR With Livial Add Back Therapy in the Management of Chronic Cyclical Pelvic Pain in Pre Menopausal Women

Start date: December 2008
Phase: Phase 4
Study type: Interventional

This is a single-centre, open-label, within patient comparison study to assess the efficacy and safety of Decapeptyl SR when administered in combination with Livial for the treatment of women with chronic cyclical pelvic pain. This will be for a 2 year period with a 6 month post treatment follow up. The study aims to recruit 40 patients.

NCT ID: NCT00710073 Completed - Clinical trials for Chronic Pelvic Pain Syndrome

Sono-Electro-Magnetic Therapy for Refractory Chronic Pelvic Pain Syndrome

Start date: May 2006
Phase: N/A
Study type: Interventional

Treatment of chronic pelvic pain syndrome (CPPS) is challenging for patients and physicians once conventional therapies fail. We hypothesize that combined sono-electro-magnetic therapy can improve refractory CPPS in men.

NCT ID: NCT00701311 Completed - Prostatitis Clinical Trials

An Open-Label Study of CC-10004 for Chronic Prostatitis/Chronic Pelvic Pain Syndrome

Start date: June 2008
Phase: Phase 2
Study type: Interventional

Prostatitis is the most common urologic diagnosis in men under the age of 50 and the third most common diagnosis in older men. In Chronic Prostatitis (CP) or Chronic Pelvic Pain Syndrome (CPPS), men have lower urinary tract symptoms, pelvic pain, sexual dysfunction and decreased quality of life. Little is known about the cause of CP/CPPS. Likewise, no definitive therapy exists for CP/CPPS. We plan to study the use of CC-10004 in men with CP/CPPS.

NCT ID: NCT00688506 Completed - Clinical trials for Chronic Pelvic Pain Syndrome

Combined Sono-electro-magnetic Therapy for Treatment of Refractory Chronic Pelvic Pain Syndrome

Start date: November 2009
Phase: N/A
Study type: Interventional

Treatment of chronic pelvic pain syndrome (CPPS) is challenging for patients and physicians once conventional therapies fail. We hypothesize that combined sono-electro-magnetic therapy can improve refractory CPPS in men. In addition, we postulate that combined sono-electro-magnetic therapy as well as placebo therapy has a significant effect on brain activity detectable by functional MRI.

NCT ID: NCT00675298 Completed - Clinical trials for Urinary Bladder, Overactive

Linkage Analysis in Interstitial Cystitis

IC
Start date: January 2006
Phase: N/A
Study type: Observational

Urologic pelvic pain syndrome (UCPPS), variably termed painful bladder syndrome/interstitial cystitis (PBS/IC) in females and chronic prostatitis/chronic pelvic pain syndrome in men (CP/CPPS), is a chronic, debilitating clinical syndrome presenting as severe pelvic pain with extreme urinary urgency and frequency in the absence of any known cause. The etiologic mechanisms underlying UCPPS are unknown, but recurrence, risks to siblings of affected individuals, concordance among monozygotic twins, and our own preliminary studies indicate a strong genetic contribution to the cause of UCPPS. The overall goal of this proposal is use novel approaches to understand the basis of UCPPS, to identify candidate genes containing mutations that result in UCPPS and determine how the different encoded proteins of these genes interact with one another in a common biological pathway. Ultimately, understanding how mutations in at least five different genes yield the symptoms of UCPPS should lead to improved diagnosis and possible therapies.