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Hepatic Encephalopathy clinical trials

View clinical trials related to Hepatic Encephalopathy.

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NCT ID: NCT01434056 Not yet recruiting - Clinical trials for Hepatic Encephalopathy

Prediction of Mortality in Patients Waiting for Liver Transplantation Through Brain Magnetic Resonance Imaging

Start date: November 2011
Phase: N/A
Study type: Observational

The purpose of this study is to determine whether hepatic encephalopathy, measured through magnetic resonance imaging, electroencephalogram and neuropsychological evaluation adds prognostic information to patients who are waiting for liver transplantation. If this model improves mortality prediction this might be used in the future for organ allocation.

NCT ID: NCT01356121 Recruiting - Clinical trials for Hepatic Encephalopathy

Trial Comparing Sedation for Endoscopy With Propofol Versus Midazolam in Cirrhotics

Start date: November 2010
Phase: Phase 4
Study type: Interventional

To compare effects of sedation for upper gastrointestinal endoscopy with propofol and midazolam on psychometric tests and critical flicker frequency (CFF) in cirrhotics

NCT ID: NCT01283152 Completed - Cirrhosis Clinical Trials

Efficacy Study of Polyethylene Glycol 3350-electrolyte Solution (GoLYTELY®) Versus Lactulose in Patients With Hepatic Encephalopathy.

PSE
Start date: January 2011
Phase: N/A
Study type: Interventional

This study is being done to find out if the laxative polyethylene glycol (also known as GoLYTELY® or Miralax®) can treat your hepatic encephalopathy (confusion due to your liver disease and/or cirrhosis) better and/or more safely than lactulose (another laxative). In this study, the investigators will evaluate if polyethylene glycol (GoLYTELY®) is more effective than lactulose on neurocognition (memory and thinking skills) and determine if it decreases the hospital stay.

NCT ID: NCT01223768 Completed - Clinical trials for Hepatic Encephalopathy (Grade 3 of the West Haven Grading Scale)

Acetyl-L-Carnitine In Severe Hepatic Encephalopathy

Start date: July 2002
Phase: N/A
Study type: Interventional

To assess the clinical efficacy of ALC in the treatment of severe hepatic encephalopathy (grade 3 of the West Haven grading scale), we performed a randomised, double blind placebo-controlled study administering ALC to cirrhotic patients, evaluating the effects on ammonia levels and performance in cognitive functions.

NCT ID: NCT01223742 Completed - Clinical trials for Hepatic Encephalopathy

Oral Acetyl-L-Carnitine Therapy Reduces Fatigue In Hepatic Encephalopathy

Start date: June 2002
Phase: N/A
Study type: Interventional

The aim of this study was to evaluate the effect of exogenous ALC on the both physical and mental fatigue in mild and moderate encephalopatic patients.

NCT ID: NCT01223729 Completed - Clinical trials for Minimal Hepatic Encephalopathy

Acetyl-L-Carnitine Reduces Depression and Improves Quality of Life in Patients With Minimal Hepatic Encephalopathy

Start date: April 2002
Phase: N/A
Study type: Interventional

Minimal hepatic encephalopathy represents a common complication present in well-compensated cirrhotic patients that impairs patients daily functioning and health-related quality of life. Acetyl-L-carnitine has been shown to be useful in improving blood ammonia and cognitive functions in cirrhotic patients with minimal hepatic encephalopathy. This study evaluated the effects of acetyl-L-carnitine treatment on health related quality of life and on depression in patients with minimal hepatic encephalopathy.

NCT ID: NCT01218568 Completed - Clinical trials for Hepatic Encephalopathy

Rifaximin Plus Lactulose Versus Lactulose Alone for the Treatment of Hepatic Encephalopathy: a Double Blind Randomized Trial

Start date: October 2010
Phase: N/A
Study type: Interventional

The study will be double blind with respect to rifaximin, and randomization will be performed using tables of computer-generated random numbers. All subjects will be followed up till the recovery of HE (primary end point) or 10 days whichever is earlier. In the Lactulose group (group A) patients will receive 30-60 ml of lactulose in 2 or 3 divided doses so that patient passes 2-3 semisoft stools per day along with placebo . In the lactulose plus rifaximin group (group B) patients will receive cap rifaximin 400mg three times a day along with lactulose. Primary endpoints will be recovery of overt HE patients who will not recover from HE after 10 days will be continued on the same treatment. Total duration of the study is 1 year.

NCT ID: NCT01178372 Recruiting - Clinical trials for Hepatic Encephalopathy

Secondary Prophylaxis of Hepatic Encephalopathy in Cirrhosis

HE
Start date: September 2008
Phase: Phase 4
Study type: Interventional

Hepatic encephalopathy (HE), a challenging complication of advanced liver disease, occurs in approximately 30-45% of patients with cirrhosis. The treatment of choice is non-absorbable disaccharides, such as lactulose and lactitol.Probiotics are effective in the treatment of minimal hepatic encephalopathy which precipitates hepatic encephalopathy. The investigators will assess the effects of lactulose and probiotics for the prevention of recurrence of HE (secondary prophylaxis) in patients after the recovery of an episode of overt hepatic encephalopathy.

NCT ID: NCT01175538 Recruiting - Clinical trials for Hepatic Encephalopathy

Primary Prophylaxis of Hepatic Encephalopathy in Patients With Cirrhosis

Start date: January 2008
Phase: Phase 4
Study type: Interventional

To assess the effects of lactulose for the prevention of first episode of altered sensorium (hepatic encephalopathy, primary prophylaxis) in patients with cirrhosis.

NCT ID: NCT01151475 Completed - Liver Cirrhosis Clinical Trials

Cerebral Oxygen, Blood Flow and Ammonia Uptake in Patients With Cirrhosis and an Acute Episode of Hepatic Encephalopathy (HE)

Start date: August 2010
Phase: N/A
Study type: Observational

We wish to study patients with liver cirrhosis during and 2 weeks after an acute episode of liver coma (hepatic encephalopathy). By means of Positron Emission Tomography we investigate; brain blood flow, brain ammonia uptake and brain oxygen consumption. We hypothesize that blood flow an oxygen consumption is diminished and ammonia uptake increased during an acute episode of hepatic encephalopathy.