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Gastrointestinal Stromal Tumors clinical trials

View clinical trials related to Gastrointestinal Stromal Tumors.

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NCT ID: NCT02336724 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor

A Study of Famitinib in Patients With Gastrointestinal Stromal Tumor

Start date: March 2012
Phase: Phase 2
Study type: Interventional

Famitinib is a tyrosin-inhibitor agent targeting at c-Kit, VEGFR2, PDGFR, VEGFR3, Flt1 and Flt3. Phase I study has shown that the toxicity is manageable. The purpose of this study is to evaluate the efficacy and safety profile of Famitinib in patients with advanced or metastatic gastrointestinal stromal tumor who failed from imatinib therapy.

NCT ID: NCT01991379 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor (GIST)

MEK162 in Combination With Imatinib Mesylate in Patients With Untreated Advanced Gastrointestinal Stromal Tumor (GIST)

Start date: November 2013
Phase: Phase 1/Phase 2
Study type: Interventional

The purpose of this study is to evaluate the effects, good and/or bad, of MEK162 and imatinib on the patient and on Gastrointestinal Stromal Tumor (GIST). Funding Source - FDA OOPD, Array/Pfizer

NCT ID: NCT01541709 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumors

Imatinib Dose Escalation to 800 mg/Day in Korean Patients With Metastatic or Unresectable GIST Harboring KIT Exon 9 Mutation: KENEDI

Start date: March 2012
Phase: Phase 2
Study type: Interventional

KIT exon 9 mutants had poorer survival compared with KIT exon 11 mutants when they were treated with the same dose of imatinib, 400 mg per day, and that patients with KIT exon 9 mutation had better progression-free survival with imatinib treatment at an escalated dose, 800 mg per day, than with imatinib treatment at a dose of 400 mg per day.10,11 Based on the results, imatinib 800 mg per day is now considered the standard dose for the treatment of patients with metastatic or unresectable GIST showing KIT exon 9 mutation in Western countries.

NCT ID: NCT01391611 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor (GIST)

Pazopanib in Imatinib Refractory or Intolerant Gastrointestinal Stromal Tumors (GIST)

Start date: July 2011
Phase: Phase 2
Study type: Interventional

This study is being done to gather information about the safety (any harmful effects) and effectiveness (usefulness) of Pazopanib in the treatment of Gastrointestinal Stroma Tumors (GIST) that cannot be treated by surgery or has spread to other organs. The Food and Drug Administration (FDA) have approved Pazopanib for the treatment of advanced kidney cancer but it is not approved for the treatment of GIST. The investigators hope to learn about the safety and usefulness (effectiveness) of Pazopanib for patients with GIST. Primary Objective: Non-progression rate based on RECIST criteria (CR+PR+SD) Secondary Objectives: - Response per Choi criteria - 6 month progression-free survival - Safety and tolerability

NCT ID: NCT01178307 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumors

Symptom Inventory for Gastrointestinal Stromal Tumors

Start date: July 29, 2010
Phase:
Study type: Observational

The goal of this research study is to better understand the symptoms experienced by patients with GIST. There are 3 parts to this study. In Part 1, participants will complete 1 set of interviews and questionnaires about GIST symptoms. In Part 2, the importance of some symptoms to patients with GIST will be rated by doctors, nurses, patients, and family caregivers. In Part 3, participants will complete questionnaires about GIST symptoms over 1 year. You are being asked to take part in Part 3 of the study.

NCT ID: NCT00756509 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumors

Treatment of Patients With Metastatic or Unresectable Gastrointestinal Stromal Tumors in First Line With Nilotinib

Start date: August 29, 2008
Phase: Phase 4
Study type: Interventional

The purpose of this multicenter, single-arm, exact binomial single-stage, phase II trial is to evaluate the efficacy of Nilotinib in patients with unresectable or metastatic gastrointestinal stromal tumors

NCT ID: NCT00685828 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor

Imatinib Mesylate in Treating Patients With Unresectable or Metastatic Gastrointestinal Stromal Tumor

Start date: January 2001
Phase: Phase 3
Study type: Interventional

RATIONALE: Imatinib mesylate may stop the growth of tumor cells by blocking some of the enzymes needed for cell growth. It is not yet known which dose of imatinib mesylate is more effective in treating gastrointestinal stromal tumor. PURPOSE: This randomized phase III trial is studying two different doses of imatinib mesylate to compare how well they work in treating patients with unresectable or metastatic gastrointestinal stromal tumor.

NCT ID: NCT00265798 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor

Sorafenib in Treating Patients With Malignant Gastrointestinal Stromal Tumor That Progressed During or After Previous Treatment With Imatinib Mesylate and Sunitinib Malate

Start date: September 14, 2005
Phase: Phase 2
Study type: Interventional

This phase II trial is studying how well sorafenib works in treating patients with malignant gastrointestinal stromal tumor that progressed during or after previous treatment with imatinib mesylate and sunitinib malate. Sorafenib may stop the growth of tumor cells by blocking some of the enzymes needed for cell growth and by blocking blood flow to the tumor.

NCT ID: NCT00112463 Active, not recruiting - Clinical trials for Gastrointestinal Stromal Tumor

Depsipeptide (Romidepsin) in Treating Patients With Metastatic or Unresectable Soft Tissue Sarcoma

Start date: January 2004
Phase: Phase 2
Study type: Interventional

This phase II trial studies how well depsipeptide (romidepsin) works in treating patients with metastatic or unresectable soft tissue sarcoma. Drugs used in chemotherapy, such as depsipeptide, work in different ways to stop the growth of tumor cells, either by killing the cells or by stopping them from dividing.