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Epidermolysis Bullosa clinical trials

View clinical trials related to Epidermolysis Bullosa.

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NCT ID: NCT03836001 Completed - Clinical trials for Epidermolysis Bullosa

A Neurokinin-1 Receptor Antagonist for the Treatment of Pruritus in Patients With Epidermolysis Bullosa

Start date: April 18, 2019
Phase: Phase 2
Study type: Interventional

To determine if Serlopitant (when taken by mouth) is safe and works on itch in patients aged 13 and above with EB.

NCT ID: NCT03786237 Recruiting - Clinical trials for Squamous Cell Carcinoma

Rigosertib for RDEB-SCC

Start date: April 12, 2021
Phase: Phase 1/Phase 2
Study type: Interventional

Epidermolysis bullosa (EB) is a heritable skin disease characterized by marked fragility of epithelialized tissue with blistering in skin and mucous membranes following the slightest mechanical trauma. Eighty percent of all patients suffering from recessive dystrophic EB (RDEB), a subtype originating from mutations in the COL7A1 gene, develop squamous cell carcinoma (SCC). In RDEB patients SCC presents early (most patients are in their 20s or 30s) and shows a highly aggressive metastatic course which often leads to premature death at this young age. In light of scarce data on the efficacy and safety of systemic treatment regimens for advanced SCC, the investigators propose to perform a small, "first in EB " trial of an experimental drug called rigosertib for the treatment of EB cancer. The trial will be conducted in two study centres, in London and Salzburg, and will last approximately 2.5 years with each patient recruited being in the study for 1 year. The drug is a polo-like kinase inhibitor interfering with different molecular pathways that are essential for cancer cell growth. Rigosertib was developed by Onconova Therapeutics and is currently tested in several clinical trials for a number of other cancers including myelodysplastic syndrome (a cancer of the blood). The investigators have identified that rigosertib most selectively kills EB cancer cells in vitro while leaving normal EB skin cells unaffected. This project will evaluate whether rigosertib is capable of inducing an anti-cancer response in EB patients and whether the drug is well-tolerated. Mechanisms of molecular targeting of squamous cancer cells by rigosertib will further be investigated in EB patients, also aiming at the identification of biomarkers that may allow the predictive identification of best responders.

NCT ID: NCT03752905 Completed - Clinical trials for Recessive Dystrophic Epidermolysis Bullosa

A Phase 1/2 Trial of PTR-01 in Adult Patients With Recessive Dystrophic Epidermolysis Bullosa (RDEB)

PTR-01-001
Start date: January 9, 2019
Phase: Phase 1/Phase 2
Study type: Interventional

Protocol PTR-01-001 is a Phase 1/2 study of PTR-01. The study is divided into an up to 4-week Screening Period, a 10-week Treatment Period and an 8-week Follow-up Period. Cohorts 1, 2, 3 and 4 will consist of 2, 4, 3 and 3 patients respectively. Each cohort will consist of patients divided into two groups (Group 1 and Group 2) randomized in a 1:1 ratio. Patients in Group 1 will receive three doses of active drug followed by 3 doses of saline control. Patients in Group 2 will receive three doses of saline control followed by 3 doses of active drug. Cohort 1 patients randomized to Group 1 will receive 3 doses of active treatment (PTR-01) at a dose of 0.1 mg/kg followed by 3 doses of saline control for a total of 6 doses. Cohort 1 patients randomized to Group 2 will receive 3 doses of saline control followed by 3 doses of active treatment (PTR-01) at a dose of 0.1 mg/kg for a total of 6 doses.

NCT ID: NCT03730584 Completed - Clinical trials for Epidermolysis Bullosa

Evaluation of the Efficacy of ROPIVACAINE in Children and Young Adults With Hereditary Epidermolysis Bullosa

EBROPI
Start date: February 27, 2017
Phase: N/A
Study type: Interventional

The purpose of this study is to determine whether topical application of Ropivacaine is effective for treating refractory pain during dressing changes and so improve quality of life of patients (newborn, child, adolescent or adults under 21) suffering from hereditary epidermal epidermolysis bullosa.

NCT ID: NCT03632265 Not yet recruiting - Clinical trials for Recessive Dystrophic Epidermolysis Bullosa

Study of EB-101 for the Treatment of Recessive Dystrophic Epidermolysis Bullosa

VITAL
Start date: October 1, 2018
Phase: Phase 3
Study type: Interventional

VITAL is an open-label, Phase 3 study of one-time surgical application of at least 35 EB-101 grafts for the treatment of eligible, chronic open wound sites in up to 15 RDEB patients. Patient wounds will be randomized to receive grafting or remain untreated (control). Up to 6 wound sites for EB 101 grafting and at least 1 untreated control wound site will be performed for each patient.

NCT ID: NCT03605069 Terminated - Clinical trials for Epidermolysis Bullosa Dystrophica, Recessive

A Double-blind, Randomized, Intra-subject Placebo-controlled, Multicenter, Multiple Dose Study, Evaluating Safety, Proof of Mechanism, Preliminary Efficacy and Systemic Exposure in Subjects With Confirmed DDEB or RDEB Diagnosis With One or More Pathogenic Mutations in Exon 73 in the COL7A1 Gene

Start date: July 2, 2018
Phase: Phase 1/Phase 2
Study type: Interventional

A double-blind, randomized, intra-subject placebo-controlled, multicenter, multiple dose study, evaluating safety, proof of mechanism, preliminary efficacy and systemic exposure in subjects with confirmed DDEB or RDEB diagnosis with one or more pathogenic mutations in exon 73 in the COL7A1 gene.

NCT ID: NCT03578029 Terminated - Clinical trials for Dystrophic Epidermolysis Bullosa

Evaluation of the Safety and Efficacy Study of RGN-137 Topical Gel for Junctional and Dystrophic Epidermolysis Bullosa

CELEB
Start date: May 22, 2019
Phase: Phase 2
Study type: Interventional

The objective of this study is to compare the efficacy and safety of RGN-137 topical gel with that of placebo gel for treatment of junctional epidermolysis bullosa (JEB) or dystrophic epidermolysis bullosa (DEB).

NCT ID: NCT03536143 Completed - Clinical trials for Dystrophic Epidermolysis Bullosa

A Phase I/II Study of KB103, a Topical HSV1-COL7, on DEB Patients

GEM-1
Start date: May 6, 2018
Phase: Phase 1/Phase 2
Study type: Interventional

This study was conducted to assess the safety and efficacy of topical Beremagene Geperpavec (KB103, HSV1-COL7) on DEB patients.

NCT ID: NCT03529877 Completed - Clinical trials for Recessive Dystrophic Epidermolysis Bullosa

Allogeneic ABCB5-positive Stem Cells for Treatment of Epidermolysis Bullosa

Start date: February 16, 2019
Phase: Phase 1/Phase 2
Study type: Interventional

The aim of this clinical trial is to investigate the efficacy (by monitoring overall improvement of EB symptoms) and safety (by monitoring adverse events) of three doses of allo-APZ2-EB administered intravenously to patients with recessive dystrophic epidermolysis bullosa (RDEB).

NCT ID: NCT03526159 Recruiting - Clinical trials for Junctional Epidermolysis Bullosa

Gentamicin for Junctional Epidermolysis Bullosa

Start date: June 1, 2018
Phase: Phase 1/Phase 2
Study type: Interventional

Herlitz junctional epidermolysis bullosa (H-JEB), an incurable, fatal, inherited skin disease, is caused by loss-of-function mutations in the LAMA3, LAMB3 or LAMC2 genes, resulting in loss of laminin 332 and poor epidermal-dermal adherence. Eighty percent of H-JEB patients have LAMB3 mutations and about 95% of these are nonsense mutations. The investigators recently demonstrated that gentamicin readily induced nonsense mutation readthrough and produced full-length laminin beta3 in several nonsense mutations tested. Importantly, the gentamicin-induced laminin beta3 restored laminin 332 assembly, secretion, and deposition into the dermal-epidermal junction (DEJ). Newly induced laminin 332 reversed abnormal H-JEB cellular phenotypes. Herein, the investigators propose the first clinical trial of gentamicin (by topical and intravenous administration) in JEB patients with nonsense mutations. The milestones will include restored laminin 332 and hemidesmosomes at the DEJ, improved wound closure, and the absence of significant gentamicin side effects.