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Brain Death clinical trials

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NCT ID: NCT01070563 Completed - Brain Dead Clinical Trials

Transcranial Doppler Ultrasonography and Brain Death

Start date: February 2009
Phase: N/A
Study type: Observational

In France the diagnosis of brain death relies on electroencephalography (EEG) or cerebral angiography. Concerning the latter a time of 6 hours is mandatory between the clinical diagnosis and the realization of the cerebral computed tomography (CT) angiography. Transcranial doppler (TCD) is not recommended in the french guidelines for the diagnosis of brain death. In other countries, some flow patterns allow to confirm the diagnosis of brain death. The hypothesis of our study is that TCD could shorten the time between the clinical diagnosis of brain death and the realization of the CT angiography.

NCT ID: NCT00998972 Completed - Clinical trials for Chronic Renal Insufficiency

N-acetyl-cysteine (NAC) and Kidney Graft Function

Start date: September 2006
Phase: Phase 3
Study type: Interventional

The goal of this study is to evaluate the effect of N-acetyl-cysteine (NAC) administration in organ donors on the kidney graft function of recipients.

NCT ID: NCT00858390 Completed - Brain Death Clinical Trials

Nutritional Status and Enteral Absorption Capability After Brain Death

HRSA Nutrition
Start date: February 2009
Phase: N/A
Study type: Interventional

The investigators propose to assess 36 donors' nutritional status using accepted parameters (prealbumin, resting energy expenditure); to assess nutrient intestinal absorption through 13Curacil breath tests; and to evaluate serum concentrations of IL-6 and TNFalpha to determine if continuing or initiating enteral feeding and nutritional supplementation is effective in restoring or maintaining nutritional parameters.

NCT ID: NCT00808691 Completed - Sepsis Clinical Trials

Microcirculation and Oxidative Stress in Critical Ill Patients in Surgical Intensive Care Unit

Start date: September 2007
Phase: N/A
Study type: Observational

As medicine advances, many lives can be saved in the intensive care unit. However, when multiple organ failure occurs, the mortality rate of patients increases dramatically. Therefore, the major goal in the intensive care unit is to prevent the occurrence of multiple organ failure. The sepsis protocol and early goal directed treatment have great effects to reduce development of multiple organ failure and to decrease the mortality rate. However, sometime the condition of patient deteriorated in spite of both the mean blood pressure and mixed venous oxygen saturation are normal. Some experts recognize that there might be microcirculatory dysfunction of tissue or organ. The dysfunction of microcirculation might due to vasoconstriction or microthrombosis. Vasoconstriction might result from systemic inflammation, reactive oxygen species, or dysfunction of synthesis of NO (nitric oxide). Microthrombosis might result from systemic inflammation, reactive oxygen species, imbalance of coagulatory system, or damage of endothelial cell. In clinical practice, the oxidative stress is related to circulatory shock, sepsis, acute lung injury, and acute respiratory distress syndrome. This study tries to investigate the relation between oxidative stress and microcirculation. Furthermore, the investigators will try to investigate the correlation between the severity of oxidative stress and microcirculatory dysfunction and the severity of disease and prognosis. The investigators hope this study will help them to figure out the picture of disease progression of patients. It may conduct further study to modulate the oxidative stress, to improve the microcirculatory function, and finally to improve the outcome of patients.

NCT ID: NCT00615225 Completed - Brain Death Clinical Trials

Brain Death: Inflammatory Response, Apoptosis, and Endotoxin Tolerance

Start date: January 2000
Phase: N/A
Study type: Observational

We hypothesized that brain death is associated with an early systemic inflammatory response, possibly combined with activation of apoptotic cell death, two events that may contribute to induce rapid organ dysfunction. In this study of brain-dead donors and controls, we assayed plasma cytokines and soluble factors, investigated plasma endotoxin levels as a triggering factor for inflammation, measured ex vivo cytokine production by blood leukocytes to determine whether immunosuppression occurred after brain death, and examined skeletal muscle biopsies to look for evidence of inflammation and increased apoptosis in peripheral tissue.

NCT ID: NCT00310401 Completed - Pulmonary Edema Clinical Trials

The Effect of Nebulized Albuterol on Donor Oxygenation

Start date: April 2007
Phase: Phase 2
Study type: Interventional

The purpose of this study is to test the effectiveness of albuterol versus placebo with the following specific aims: a) Treatment of brain dead organ donors with albuterol will reduce pulmonary edema, improve donor oxygenation, and increase the number of lungs available for transplantation, b) Developing a blood test to predict the development of primary graft dysfunction in lung transplant recipients, and c) treating brain dead organ donors with albuterol will decrease markers of primary graft dysfunction and lead to improved lung transplant recipient outcomes and to higher rates of lungs suitable for transplantation.

NCT ID: NCT00238030 Completed - Brain Death Clinical Trials

Thyroxine Replacement in Organ Donors

Start date: December 2004
Phase: Phase 1/Phase 2
Study type: Interventional

To compare oral versus intravenous administration of thyroid hormone: 1) for reversibility of hemodynamic instability in organ donors, and, 2) the pharmacokinetics of oral vs iv thyroid administration