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Alcoholic Intoxication clinical trials

View clinical trials related to Alcoholic Intoxication.

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NCT ID: NCT04564599 Active, not recruiting - Alcohol Drinking Clinical Trials

The Rise of Ride Sharing Companies and Trends in Impaired Driving Accidents

Start date: July 6, 2023
Phase:
Study type: Observational

This will be a retrospective study with data collected from the trauma registry. We plan to complete the data collection and analysis by 12/31/2020. Data on ride sharing will be obtained from the Uber and Lyft websites. Data pertaining to number of alcohol- and drug-related motor vehicle (and auto-ped) collisions will be obtained from the Texas Department of Transportation website, the National Highway Traffic Safety Administration, the Shared-Use Mobility Center (SUMC) and the Transformation of Public Transit, the Texas A&M Transportation Institute, Texas Department of Public Safety, and the U.S. Department of Transportation website (or equivalent). Sexual assault data will be obtained as available the Sexual Assault Nurse Examiner (SANE) database as well as from Turning Point Rape Crisis Center and surrounding hospitals in the Dallas area as well as the Uber report for sexual assaults.

NCT ID: NCT03467191 Active, not recruiting - Clinical trials for Alcohol Use Disorder

Behavioral Alcohol Responses (BAR) Study

Start date: February 13, 2018
Phase: Early Phase 1
Study type: Interventional

This study aims to identify risk factors that prospectively predict alcohol problems in young adults.

NCT ID: NCT01350089 Active, not recruiting - Clinical trials for Perceived Alcohol Intoxication

Possible Effects of Energy Drink Ingestion on Perceived Alcohol Intoxication

Start date: January 2011
Phase: N/A
Study type: Interventional

The purpose of this study is to determine whether the ingestion of caffeine, Energy Drinks has an effect on perceived alcohol intoxication.

NCT ID: NCT00608686 Active, not recruiting - Clinical trials for Alcohol Intoxication

Neurosteroids and Acute Alcohol Intoxication in Humans

Start date: May 2004
Phase: Phase 1
Study type: Interventional

1. The major aims are to assess: (1) the relationship of basal and alcohol-induced neurosteroid and GABA levels to the degree of acute alcohol intoxication in healthy male and female volunteers; and (2) the effect of acute pregnenolone administration on the degree of acute alcohol intoxication in these same volunteers. Specific hypotheses are: - Baseline serum levels of pregnenolone, pregnenolone sulfate (PS), dehydroepiandrosterone (DHEA) and DHEA sulfate (DHEA-S) will be inversely correlated with the magnitude of acute behavioral responses to alcohol (sedation, anxiolysis, amnesia, psychomotor impairment and intoxication). That is, higher baseline levels of these neurosteroids will be associated with lessened behavioral responses to alcohol. - Baseline serum levels of allopregnanolone, tetrahydrodeoxycorticosterone (THDOC), androstanediol, androsterone and GABA will be directly correlated with the magnitude of acute behavioral responses to alcohol. That is, higher baseline levels of these substances will be associated with heightened behavioral responses to alcohol. - Acute alcohol ingestion, compared to placebo ingestion, will increase serum levels of allopregnanolone and THDOC and plasma levels of GABA and will decrease plasma levels of PS. (Effects on levels of other neurosteroids are not specifically predicted based on animal data but will be examined in an exploratory manner.) - Acute alcohol-induced increases in serum levels of allopregnanolone and THDOC and in plasma levels of GABA will be directly correlated with the magnitude of acute behavioral responses to alcohol. Acute alcohol-induced decreases in serum levels of PS will be directly correlated with the magnitude of acute behavioral responses to alcohol. Correlations between alcohol-induced changes in other neurosteroids and changes in behavior are not specifically predicted but will be examined in an exploratory manner. - Pregnenolone, compared to placebo, pre-treatment will antagonize the acute effects of alcohol on the behavioral measures.