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Age-Related Macular Degeneration clinical trials

View clinical trials related to Age-Related Macular Degeneration.

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NCT ID: NCT01327222 Completed - Clinical trials for Age Related Macular Degeneration

Intravitreal Bevacizumab for Low Vision in Neovascular Age-related Macular Degeneration (AMD)

LOW-VISION
Start date: February 2011
Phase: Phase 3
Study type: Interventional

The purpose of this study is to compare the effectiveness of intravitreal bevacizumab injection (IVBI) with respect to simple observation in patients with subfoveal choroidal neovascularisation (CNV) secondary to age-related macular degeneration (AMD) presenting at baseline with best corrected visual acuity (BCVA) less than 20/200.

NCT ID: NCT01319188 Terminated - Clinical trials for Coronary Artery Disease

Ranibizumab and the Risk of Arterial Thromboembolic Events

RATE
Start date: June 2010
Phase: Phase 4
Study type: Interventional

The investigators assume that ranibizumab might be dangerous in patients with history of coronary artery disease or cerebrovascular events. The main objective of study is to reveal contraindications for ranibizumab prescription in patients with history of coronary artery disease and cerebrovascular events. Moreover, an association between management with ranibizumab and ATE rate in healthy above 50 years old persons is a concern of great interest as well.

NCT ID: NCT01316198 Completed - Clinical trials for Age-Related Macular Degeneration

Effects of Xanthophylls on Optical Density

Start date: June 2010
Phase: N/A
Study type: Interventional

The objective of the study was to investigate the influence of a intervention with lutein und zeaxanthin from crucifers on the optical density of the macular pigment of patients with non exudative age-related maculopathy. It is hypothesized that the applied study beverage elevates plasma concentrations of the administered xanthophylls and the optical density after 4 weeks of intervention.

NCT ID: NCT01301443 Completed - Clinical trials for Age Related Macular Degeneration

Phase I Dose Escalation Safety Study of RetinoStat in Advanced Age-Related Macular Degeneration (AMD)

GEM
Start date: February 2011
Phase: Phase 1
Study type: Interventional

The purpose of this first in man study is to examine the safety of an experimental gene transfer agent, RetinoStat, designed to treat neovascular age-related macular degeneration.

NCT ID: NCT01298674 Completed - Clinical trials for Age-Related Macular Degeneration

Epidemiological Study of the Prevalence of Age-Related Macular Degeneration in Portugal

Start date: August 2009
Phase: N/A
Study type: Observational

This is an epidemiological study to assess the the prevalence of Age-Related Macular (ARM) Degeneration in Portugal.

NCT ID: NCT01272076 Completed - Clinical trials for Age Related Macular Degeneration

Cirrus HD-OCT Measurement of Area of Increased Light Penetration Under the Retinal Pigment Epithelium (RPE)

Start date: January 2011
Phase: N/A
Study type: Observational

The objective of this study is to compare Cirrus HD-OCT automated measurements of the illumination area under the retinal pigment epithelium (RPE) to expert manual measurements of areas of hypofluorescence typical of geographic atrophy in fundus autofluorescence (FAF) images.

NCT ID: NCT01271270 Completed - Clinical trials for Age-Related Macular Degeneration

Palomid 529 in Patients With Neovascular Age-Related Macular Degeneration

Start date: December 20, 2010
Phase: Phase 1
Study type: Interventional

Background: - Wet age-related macular degeneration (AMD) occurs when abnormal blood vessels grow in the back of the eye, and leak blood and other fluids that damage the eye, produce scarring, and lead to blindness. People diagnosed with wet AMD have increased production of a body chemical called vascular endothelial growth factor (VEGF). VEGF is important in the formation of blood vessels in the body, and decreasing the production of VEGF is believed to help wet AMD patients by preventing or slowing the growth of the abnormal blood vessels. Anti-VEGF drugs have been used to decrease the production of VEGF, but some people do not respond completely to these drugs. - A protein in the body called mTOR also plays a critical role in regulating how cells divide and grow and obtain their blood supply. The experimental chemical Palomid 529 inhibits the production of mTOR. Researchers are interested in determining whether Palomid 529 is safe and can help individuals with wet AMD who have not completely responded to anti-VEGF treatments. Objectives: - To evaluate the safety and effectiveness of Palomid 529 as a treatment for wet age-related macular degeneration in individuals who have not responded to standard anti-VEGF treatments. Eligibility: - Individuals with wet age-related macular degeneration in at least one eye that has not responded to standard anti-VEGF treatments. Design: - Prior to the first visit, participants should have been seen at the National Eye Institute clinic under a screening or teaching protocol, or NIH protocol 08-EI-0103, High Speed Indocyanine Green Angiography Findings in Induction Regimen of Intravitreal Ranibizumab Injection for Neovascular Age Related Macular Degeneration. One eye will be designated as the study eye to receive the Palomid 529 treatment. - Participants will have a full physical examination and medical history, a full eye examination to evaluate eye health and vision, angiography to examine the blood vessels in the eyes, and blood and urine tests during the study - Participants will receive an injection of Palomid 529 into the study eye every 4 weeks during the study, for a total of three injections. Participants may also receive anti-VEGF injections such as ranibizumab (Lucentis ) or bevacizumab (Avastin ) in the study eye 12 days before and 12 days after the Palomid 529 injection. - Participants may have standard-of-care treatments for the non-study eye if it has wet AMD as well, but may not receive experimental treatments in the non-study eye while they are in this study. - Participants will return for long-term follow-up examinations as directed by the study researchers.

NCT ID: NCT01269151 Completed - Clinical trials for Age-Related Macular Degeneration

Ranibizumab Short- and Log-term Effects on Retinal Function in wAMD

LucERG
Start date: October 2010
Phase: N/A
Study type: Interventional

Multifocal-electroretinoram (ERG) and microperimetry are objective criteria to asses retinal function. No information besides visual acuity exist for the development of retinal function during the course of wet AMD during therapy with Lucentis. The aim of our study is to evaluate the value of multifocal-ERG and microperimetry as an sensitive tool and early predictor of recurrence of the disease. The second goal will be to evaluate the neuroprotective effects of an adequate therapy on retinal function using microperimetry and multifocal-ERG during the course of wet AMD.

NCT ID: NCT01256827 Completed - Clinical trials for Age-related Macular Degeneration

Seven Year Update of Macular Degeneration Patients

SEVEN UP
Start date: December 2010
Phase: N/A
Study type: Observational

The purpose is to assess long term vision outcomes and disease status in patients with age-related macular degeneration previously treated with ranibizumab as participants in the Phase 3 ANCHOR and MARINA Studies and the HORIZON Study.

NCT ID: NCT01255774 Recruiting - Clinical trials for Age Related Macular Degeneration

Serial Retinal Thickness Changes And Scotoma Size Measured With Lucentis Therapy

FVF4193s
Start date: June 2007
Phase: Phase 3
Study type: Interventional

The rationale for evaluating LucentisTM (a recombinant humanized anti-human Vascular Endothelial Growth Factor (VEGF) monoclonal antibody (Fab) in patients with neovascular (wet) Age related Macular Degeneration (AMD) is based on the strong correlation between VEGF levels in aqueous and vitreous humor and active blood vessel formation. LucentisTM has been shown to maintain or improve vision in wet AMD. Since the treatments do not halt the loss of visual acuity in wet AMD, it is important to accurately measure anatomic response to treatments thereby enhancing the ability to understand patterns of response and predict the necessity for continued therapy.