Subarachnoid Hemorrhage Clinical Trial
Official title:
Searching a Dysfunction of Corticotropic and Thyrotropin Axis During the Acute Phase ( ≤48h ) of a Subarachnoid Hemorrhage Secondary to Spontaneous Rupture of Cerebral Aneurysm: Impact on the Incidence of Complications and Recovery at 1 Month
Searching a dysfunction of corticotropic and thyrotropin axis during the acute phase ( ≤48h )
of a subarachnoid hemorrhage secondary to spontaneous rupture of cerebral aneurysm. Impact on
the incidence of complications and recovery are evaluated at 1 month.
Blood sample are made within 48 hours of the onset of bleeding with assay of total plasma
cortisol, plasma ACTH at 8 am and thyroid hormones (T3, free T4 , and TSH). Dynamic test ACTH
stimulation (test Synacthene) with renewal of serum cortisol to H + 1 (60min). Evaluation in
the first 30 days of the incidence of rebleeding, hydrocephalus, of vasospasm, infection and
epilepsy. GOS to 1 month.
Searching a dysfunction of corticotropic and thyrotropin axis during the acute phase ( ≤48h )
of a subarachnoid hemorrhage secondary to spontaneous rupture of cerebral aneurysm. Impact on
the incidence of complications and recovery are evaluated at 1 month.
Blood sample are made within 48 hours of the onset of bleeding with assay of total plasma
cortisol, plasma ACTH at 8 am and thyroid hormones (T3, free T4 , and TSH). Dynamic test ACTH
stimulation (test Synacthene) with renewal of serum cortisol to H + 1 (60min). Evaluation in
the first 30 days of the incidence of rebleeding, hydrocephalus, of vasospasm, infection and
epilepsy. GOS to 1 month.
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