Clinical Trials Logo

Peripheral Arterial Disease clinical trials

View clinical trials related to Peripheral Arterial Disease.

Filter by:

NCT ID: NCT03058913 Recruiting - Clinical trials for Peripheral Arterial Disease

OEIS Outcome Data Registry

Start date: February 1, 2017
Phase:
Study type: Observational [Patient Registry]

This registry is created under the sponsorship of the Outpatient Endovascular and Interventional Society (OEIS) to monitor the clinical course and treatment outcomes of patients undergoing office-based endovascular procedures.

NCT ID: NCT03054519 Recruiting - Clinical trials for Peripheral Artery Disease

Improve PAD PERformance With METformin

PERMET
Start date: May 1, 2017
Phase: Phase 3
Study type: Interventional

The PERMET trial will determine whether metformin daily for six months improves six-minute walk performance in individuals with peripheral artery disease compared to placebo.

NCT ID: NCT03048890 Recruiting - Clinical trials for Peripheral Artery Disease

VascTrac Peripheral Arterial Disease (PAD) Study

VascTrac
Start date: March 1, 2017
Phase:
Study type: Observational

VascTrac is a mobile medical application that tracks users' physical activity levels in order to predict endovascular failure of patients with Peripheral Artery Disease.

NCT ID: NCT02965677 Recruiting - Clinical trials for Peripheral Arterial Disease

A Study of The Femoral Popliteal Artery Treated With LEGFLOW OTW

Start date: December 2016
Phase: N/A
Study type: Interventional

To evaluate the safety and efficacy of the Paclitaxel Releasing Peripheral Balloon Dilatation Catheter (LEGFLOW) compared with the standard balloon (Admiral Xtreme) for the treatment of stenosis or occlusions in femoral popliteal artery.

NCT ID: NCT02962232 Recruiting - Clinical trials for Peripheral Artery Disease

A Study of Below The Knee Arteries' Stenosis or Occlusion Treated With LEGFLOW OTW

Start date: November 2016
Phase: N/A
Study type: Interventional

To evaluate the safety and efficacy of the Paclitaxel Releasing Peripheral Balloon Dilatation Catheter (LEGFLOW) compared to the PTA catheter (AMPHIRION DEEP) in treatment of stenosis or occlusion in below the knee artery.

NCT ID: NCT02959606 Recruiting - Clinical trials for Peripheral Arterial Disease

SAFE (Sarpogrelate Anplone in Femoro-popliteal Artery Intervention Efficacy) Study

Start date: December 2016
Phase: Phase 4
Study type: Interventional

After endovascular treatment (EVT) for peripheral artery disease (PAD), dual antiplatelet therapy (DAAT) of aspirin (ASA) and clopidogrel are currently drug of choice to prevent occlusion. Anplone SR®, controlled-released Sarpogrelate hydrochloride, has been introduced as an anti-platelet agent for the drug of PAD. The aim of this study was to compare the efficacy and safety of Anplone + aspirin and clopidogrel + aspirin in patients who underwent EVT for femoro-popliteal occlusive disease.

NCT ID: NCT02915796 Recruiting - Clinical trials for Peripheral Arterial Disease

Autologous CD133(+) Cells as an Adjuvant to Below the Knee Percutaneous Transluminal Angioplasty

Start date: September 2016
Phase: Phase 1
Study type: Interventional

The main aim of the present study was to evaluate the therapeutic potential and safety of transarterial infusion of granulocyte colony stimulating factor (G-CSF) mobilized cluster of differentiation (CD) 133(+) cells when combined with percutaneous transluminal angioplasty (PTA) in treatment of below the knee (BTK) peripheral arterial disease (PAD) in diabetic patients.

NCT ID: NCT02877563 Recruiting - Clinical trials for Peripheral Artery Disease

Contrast Ultrasound Assessment of Perfusion Changes After Peripheral Artery Revascularization

Start date: June 1, 2018
Phase:
Study type: Observational

This study represents an important step to validate the use of contrast-enhanced ultrasound (CEU) as a clinical research tool that can be used as a biologic readout of new therapies for Peripheral Artery Disease (PAD), and as a clinically-reasonable method for assessing impact of revascularization. The primary aim is to establish that CEU limb perfusion imaging can be used to accurately assess therapeutic improvements in tissue perfusion that are linked to symptom improvement.

NCT ID: NCT02850432 Recruiting - Clinical trials for Peripheral Arterial Disease

Clinical and MRI Cardiac Biomarkers on Peripheral Arterial Disease in Patients With Long Term DM

CMRPADDM
Start date: May 1, 2016
Phase: N/A
Study type: Interventional

The aim of this study is to establish a clinically feasible simultaneous evaluation of heart and lower limb using MRI and to assess the combined benefit of clinical and cardiac MRI imaging markers in the evaluation of PAD and prediction of treatment outcome.

NCT ID: NCT02842424 Recruiting - Clinical trials for Peripheral Arterial Disease

Ramipril Treatment of Claudication: Oxidative Damage and Muscle Fibrosis

Start date: February 25, 2016
Phase: Phase 4
Study type: Interventional

Peripheral artery disease (PAD) is a manifestation of atherosclerosis that produces progressive narrowing and occlusion of the arteries supplying the lower extremities. The most common clinical manifestation of PAD is claudication, i.e., a severe functional limitation identified as gait dysfunction and walking-induced leg muscle pain relieved by rest. The standard therapies for claudication include the medications cilostazol and pentoxifylline, supervised exercise therapy and operative revascularization. Recent data demonstrated that 24 weeks of treatment with the angiotensin-converting enzyme (ACE) inhibitor Ramipril produces improvements in the walking performance of patients with claudication that are higher than those of cilostazol and pentoxifylline and similar to those produced by supervised exercise therapy and operative revascularization. The mechanisms by which Ramipril therapy produces this impressive improvement in the functional capacity of claudicating patients remain unknown. The Investigators hypothesize that treatment of claudicating PAD patients with Ramipril will improve walking performance and quality of life by improving the myopathy of the gastrocnemius. Improved myopathy is a consequence of reduced oxidative damage, reduced TGF-β1 production by vascular smooth muscle cells and reduced collagen deposition in the affected gastrocnemius.