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Schizoaffective Disorder clinical trials

View clinical trials related to Schizoaffective Disorder.

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NCT ID: NCT00057135 Completed - Schizophrenia Clinical Trials

Improving Antipsychotic Adherence Among Patients With Serious Mental Illness

Start date: November 2002
Phase: N/A
Study type: Interventional

This is a randomized controlled trial examining the effectiveness of a pharmacy-based intervention designed to improve adherence with antipsychotic medications among patients with serious mental illness.

NCT ID: NCT00051233 Completed - Schizophrenia Clinical Trials

Treatment of Schizophrenia Through Internet-Based Psychoeducation

Start date: January 2003
Phase: Phase 2
Study type: Interventional

The purpose of this study is to assess the effectiveness of a Web-based psychoeducational program in helping people with schizophrenia and their families manage the disease.

NCT ID: NCT00036361 Completed - Schizophrenia Clinical Trials

Broad Effectiveness: Study With Aripiprazole

Start date: July 2002
Phase: Phase 3
Study type: Interventional

The purpose of this study is to learn if aripiprazole is effective in the treatment of a large number of persons diagnosed with schizophrenia or schizoaffective disorders

NCT ID: NCT00034801 Completed - Depression Clinical Trials

Olanzapine Versus Active Comparator in the Treatment of Depression in Patients With Schizophrenia

Start date: September 2001
Phase: Phase 4
Study type: Interventional

This is a research study comparing the safety and efficacy of two active study medications

NCT ID: NCT00018642 Completed - Schizophrenia Clinical Trials

Quetiapine vs Haloperidol Decanoate for the Long Term Treatment of Schizophrenia and Schizoaffective Disorder

Start date: April 1997
Phase: N/A
Study type: Interventional

The purpose of this research study is to determine whether a new drug for schizophrenia is better for the maintenance treatment than a standard drugs currently prescribed. The new medication is called quetiapine and it will be compared with a standard medication called haloperidol decanoate. The study will determine if quetiapine causes fewer problems than haloperidol with side effects such as stiffness and restlessness and whether it costs the VA more or less to treat patients with quetiapine. In addition, blood samples will be collected every three months to determine if certain chemicals in the blood can influence the outcome of the subjects' illness.

NCT ID: NCT00018629 Completed - Schizophrenia Clinical Trials

Cognitive - Behavioral Therapy

Start date: May 2000
Phase: Phase 2
Study type: Interventional

The purpose of this study is to determine if Cognitive - Behavioral Social Skills Training (CBSST) improves functioning in older patients with schizophrenia.

NCT ID: NCT00007774 Completed - Schizophrenia Clinical Trials

To Determine if Olanzapine is More Cost Effective Than Haloperidol for the Treatment of Schizophrenia

Start date: March 1998
Phase: Phase 4
Study type: Interventional

Although currently marketed antipsychotic drugs are useful in the treatment of schizophrenia, efficacy and safety profiles need to be improved. Forty to eighty percent of patients either fail to respond or only partially respond to conventional antipsychotic agents. Secondary symptoms may be unimproved even in patients who respond to treatment. A variety of adverse events occur in patients receiving currently available agents. The severity of these events contributes to the poor compliance that is observed in this patient population. Olanzapine is a novel antipsychotic agent with a reduced incidence of extrapyramidal symptoms. Other side effects are minimal.

NCT ID: NCT00001486 Recruiting - Schizophrenia Clinical Trials

Genetic Study of Schizophrenia

Start date: July 15, 1995
Phase:
Study type: Observational

This large ongoing study at NIMH investigates the neurobiology of schizophrenia by identifying susceptibility genes, evaluating their impact on brain function to better understand how to treat and prevent this illness.

NCT ID: NCT00001198 Terminated - Schizophrenia Clinical Trials

Evaluating Genetic Risk Factors for Childhood-Onset Schizophrenia

Start date: March 19, 1984
Phase: N/A
Study type: Observational

A study of children and adolescents (current N=100) with very early onset by age 12 (COS) of DSM-III-R defined schizophrenia with (97-M-0126) is examining the clinical, neurobiological, early neurodevelopmental, genetic, and clinical drug response characteristics of these cases. Earlier studies have documented the continuity between COS and adult onset cases (See Jacobsen and Rapoport, 1998 for review). The focus has now shifted to increasing the sample size and evaluation of familial risk factors including: psychiatric diagnoses of family members; smooth pursuit eye movements; neuropsychological tests deficits, and obtaining blood for cell lines for genetic studies (family members only, this is also covered under 96-M-0060, Dr. Ellen Sidransky). A study of obstetrical records of COS probands indicated no increase in adverse pre or perinatal events for these cases compared with obstetrical records of their siblings (Nicolson et al submitted). In contrast, several findings point to increased risk for these probands. To date, a total of 5 (10.4%) COS subjects were found to have previously unknown cytogenetic abnormalities (Microdeletion of 22q11 (3 cases), (Usiskin et al, submitted), Mosaic 45X0 (one case) (Kumra et al, 1998) and balanced 1:7 translocation (Gordon et al 1994). The study of first degree relatives of these very rare cases addresses the hypothesis that risk factors, most probably genetic, are increased in immediate family members relative both to community controls and to the relatives of patients with chronic, treatment resistant, adult-onset schizophrenia (AOS). A second hypothesis is that COS familial risk factors show significant relationship to the developmental delays/abnormalities being observed in the COS probands. As a total of 50 additional COS subjects will be studied over the next 5 years, the pediatric control sample for the probands will also be increased, determined by the need to have concurrent measures for patients and controls to maintain measurement validity. Thus a total of 600 additional subjects are to be studied including 50 controls for COS probands, 150 COS relatives, 150 controls for COS relatives, and 250 relatives of adult onset schizophrenics (AOS).