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Scleroderma, Systemic clinical trials

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NCT ID: NCT02260557 Completed - Clinical trials for Raynaud's Phenomenon Secondary to Systemic Sclerosis

Effects of Selexipag in Adults With Raynaud's Phenomenon Secondary to Systemic Sclerosis

Start date: October 2014
Phase: Phase 2
Study type: Interventional

The primary objective of the study is to determine the activity of selexipag on Raynaud attack frequency in subjects with Raynaud's Phenomenon (RP) secondary to Systemic Sclerosis (SSc).

NCT ID: NCT02228850 Completed - Clinical trials for Raynaud's Phenomenon Secondary to Systemic Sclerosis

Study of Acute Peripheral Vascular Effects, Safety and Tolerability in Subjects With Raynaud's Phenomenon Secondary to Systemic Sclerosis

Start date: November 2014
Phase: Phase 2
Study type: Interventional

The purpose of this study is to evaluate pharmacodynamics (PD) in adult subjects with a diagnosis of Raynaud's Phenomenon secondary to Systemic Sclerosis (SSc).

NCT ID: NCT02213705 Completed - ADULT Clinical Trials

Treatment of Refractory Sever Systemic Scleroderma by Injection of Allogeneic Mesenchymal Stem Cells

MSC
Start date: May 6, 2014
Phase: Phase 1/Phase 2
Study type: Interventional

The main ailm of this phase I-II study is to evaluate toxicity and efficacy of allogenic mesenchymal stem cell therapy to treat severe systemic sclerosis. In practice this treatment will be given to patients with a rapidly evolutive disease or refractory to cyclophosphamide.

NCT ID: NCT02212249 Completed - Systemic Sclerosis Clinical Trials

Soluble VE-cadherin and VE-cadherin Antibody in Sclerodermic Sclerosis

Sclerocadh1
Start date: May 2014
Phase: N/A
Study type: Interventional

Ve-cadherin is expressed in endothelial cells. Systemic slerosis is a rare auto-immune disease with a endothelial dysfunction. This study is to evaluated the level of soluble VE-cadherin and VE-cadherin antibody in patients with systemic slerosis.

NCT ID: NCT02161406 Completed - Clinical trials for Diffuse Cutaneous Systemic Sclerosis

A Study of Subcutaneous Abatacept to Treat Diffuse Cutaneous Systemic Sclerosis

ASSET
Start date: September 2014
Phase: Phase 2
Study type: Interventional

The study hypothesis is that SC abatacept is safe and shows evidence of efficacy (improvement in modified Rodnan score [mRSS]) in patients with diffuse cutaneous systemic sclerosis (dcScc) compared to matching placebo.

NCT ID: NCT02105818 Completed - Systemic Sclerosis Clinical Trials

Swallowing Difficulties With Medication Intake and Coping Strategies in Patients With Systemic Sclerosis

SWAMECO
Start date: March 2014
Phase: N/A
Study type: Observational

The purpose of this study is the validation of a newly developed self-report questionnaire which aims at determining the prevalence, location and intensity of SWAllowing difficulties with drug intake, and describing the impact on MEdication regimen focusing on COping strategies (SWAMECO).

NCT ID: NCT02062125 Completed - Scleroderma Clinical Trials

Calcinosis in a Single-Center Scleroderma Population

SSc-calcinosis
Start date: October 31, 2012
Phase:
Study type: Observational [Patient Registry]

This is a 10 year study of scleroderma patients with calcinosis 1) to better understand how common and if there are any risk factors for having calcinosis 2) to identify common complications associated with scleroderma-related calcinosis. .

NCT ID: NCT02047708 Completed - Clinical trials for Chronic Kidney Disease

Zibotentan Better Renal Scleroderma Outcome Study

ZEBRA
Start date: October 2014
Phase: Phase 2
Study type: Interventional

Many patients with scleroderma have damage to their kidneys caused by the disease. There is limited evidence for treatments to prevent this damage or stop it progressing. Blocking a substance in the blood called endothelin has helped treat some aspects of scleroderma. The purpose of this study is to see how effective a new endothelin blocker called Zibotentan is in treating patients who have scleroderma and have gone on to develop reduced kidney function as a complication. It will be given in addition to the accepted treatments used for scleroderma. There will be three parts to this study each for a different group of patients: - ZEBRA 1 for patients with mild or moderate kidney disease caused by scleroderma - ZEBRA 2A for patients with a more severe, acute form of kidney disease caused by scleroderma (scleroderma renal crisis) who do not require dialysis - ZEBRA 2B for patients who have had scleroderma renal crisis and are on dialysis

NCT ID: NCT02006420 Completed - Scleroderma Clinical Trials

Scleroderma ARFI Imaging of the Skin

ARFI
Start date: December 2013
Phase:
Study type: Observational

Scleroderma and other rheumatologic conditions can affect the skin. Scleroderma in particular involves skin thickening and hardening. Currently, looking at the degree that the skin is affected by scleroderma is measured based on a combination of a physical exam and a skin biopsy. The researchers propose to measure skin hardness using ultrasound imaging of elasticity. They will use a technique using acoustic radiation force impulse/shear wave velocity imaging , known as ARFI/SVI). The investigators hypothesize that ARFI/SVI may be able to distinguish between normal skin and skin affected by scleroderma.. This tool may also help to quantify the amount of fibrosis in the skin. This type of radiologic biomarker could be used to help confirm the diagnosis of scleroderma.

NCT ID: NCT01988506 Completed - Clinical trials for Rheumatoid Arthritis

Induction of Regulatory t Cells by Low Dose il2 in Autoimmune and Inflammatory Diseases

TRANSREG
Start date: January 6, 2014
Phase: Phase 2
Study type: Interventional

TRANSREG will assess the safety and biological efficacy of low-dose IL2 as a Treg inducer in a set of 14 autoimmune and auto-inflammatory diseases, with the aim to select diseases in which further therapeutic development will be performed. Extensive biological- and immune-monitoring pre- and post-IL2 will contribute (i) to define the common or distinct processes responsible for the breakdown of immunological tolerance in these pathologies and (ii) to discover potential biomarkers of the IL2 response.